Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Consequences of adaptation of TAL effectors on host susceptibility to Xanthomonas.

PLoS genetics - 1 Jan 2021

Teper Doron, Wang Nian

Abstract excerpt

Transcription activator-like effectors (TALEs) are virulence factors of Xanthomonas that induce the expression of host susceptibility (S) genes by specifically binding to effector binding elements (EBEs) in their promoter regions. The DNA binding specificity of TALEs is dictated by their tandem repeat regions, which are highly variable between different TALEs. Mutation of the EBEs of S genes is being utilized as...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Proteins
Share to Citrus
Share to Gene Expression Regulation, Plant

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Host-Pathogen Interactions

Share to Plant Diseases

Share to Plant Proteins

Share to Virulence Factors

Share to Xanthomonas

Source

Open in PubMed Open full text

Identifiers

PMID: 33465093
DOI: 10.1371/journal.pgen.1009310
PMCID: PMC7845958

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Consequences of adaptation of TAL effectors on host susceptibility to Xanthomonas.PMID 33465093Read ↗

An engineered promoter driving expression of a microbial avirulence gene confers recognition of TAL effectors and reduces growth of diverse Xanthomonas strains in citrus.Semantic proximity Read ↗

Lateral organ boundaries 1 is a disease susceptibility gene for citrus bacterial canker disease.Semantic proximity Read ↗

Five phylogenetically close rice SWEET genes confer TAL effector-mediated susceptibility to Xanthomonas oryzae pv. oryzae.Semantic proximity Read ↗

The TALE effector PthA4 of <i>Xanthomonas citri</i> subsp. <i>citri</i> indirectly activates an expansin gene <i>CsEXP2</i> and an endoglucanase <i>CsEG1</i> via CsLOB1 to cause citrus canker symptomsSemantic proximity Read ↗

Activation of three targets by a TAL effector confers susceptibility to bacterial blight of cottonSemantic proximity Read ↗

Variations in type III effector repertoires, pathological phenotypes and host range of Xanthomonas citri pv. citri pathotypes.Semantic proximity Read ↗

Activation of three targets by a TAL effector confers susceptibility to bacterial blight of cottonSemantic proximity Read ↗

Comparative transcriptional analyzes of Xanthomonas citri subsp. citri reveal mechanisms of adaptation and bacterial virulence in the early stage of citrus canker diseaseSemantic proximity Read ↗

A TAL effector repeat architecture for frameshift binding.Semantic proximity Read ↗

Two TAL effectors of <i>Xanthomonas citri</i> pv. <i>malvacearum</i> target <i>GhSWEET15</i> as the susceptibility genes for bacterial blight of cottonSemantic proximity Read ↗

Genomic insights into the TAL effector diversity and SWEET gene targeting in Indian strains of Xanthomonas oryzae pv. oryzae.Semantic proximity Read ↗

A strain of an emerging Indian pathotype of <i>Xanthomonas oryzae</i> pv. oryzae defeats the rice bacterial blight resistance gene <i>xa13</i> without inducing a clade III <i>SWEET</i> gene and is nearly identical to a recent Thai isolateSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.