Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Dynamic Transcriptome Reveals the Mechanism of Liver Injury Caused by DHAV-3 Infection in Pekin Duck

6 Nov 2020

Abstract excerpt

Duck hepatitis A virus 3 (DHAV-3) is a wild endemic virus, which seriously endangers the duck industry in China. The present study aims to elucidate the mechanism of duck resistance to DHAV-3 infection. Both resistant and susceptible ducks were challenged with DHAV-3, and the dynamics of liver histopathological features and transcriptome were analyzed at 6, 12, 15, and 24 h post-infection (hpi). The...

Topics

Join the communities discussing this publication.

Animal Disease Management and Epidemiology
interferon and immune responses
Viral Infections and Immunology Research

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 33240261
DOI: 10.3389/fimmu.2020.568565

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Dynamic Transcriptome Reveals the Mechanism of Liver Injury Caused by DHAV-3 Infection in Pekin DuckPMID 33240261Read ↗

Comparative transcriptome reveals the effect of IFITM1 on differential resistance to duck hepatitis A virus genotype 3 in Pekin ducks.Semantic proximity Read ↗

Whole-transcriptome sequencing revealed the role of noncoding RNAs in susceptibility and resistance of Pekin ducks to DHAV-3.Semantic proximity Read ↗

Plasma lipidome reveals susceptibility and resistance of Pekin ducks to DHAV-3.Semantic proximity Read ↗

Proteomics reveals the effect of type I interferon on the pathogenicity of duck hepatitis A virus genotype 3 in Pekin ducks.Semantic proximity Read ↗

Effect of duck hepatitis A virus genotype 3 infection on glucose metabolism of Pekin ducklings and underlying mechanism.Semantic proximity Read ↗

NOD1 Is Associated With the Susceptibility of Pekin Duck Flock to Duck Hepatitis A Virus Genotype 3.Semantic proximity Read ↗

Comparative Transcriptomic Analysis of Immune-Related Gene Expression in Duck Embryo Fibroblasts Following Duck Tembusu Virus InfectionSemantic proximity Read ↗

Pathogenicity of duck hepatitis A virus type 3 and innate immune responses of the ducklings to virulent DHAV-3Semantic proximity Read ↗

Advances in the Duck Hepatitis A virus and lessons learned from those in recent years.Semantic proximity Read ↗

Host Differences Affecting Resistance and Susceptibility of the Second Generation of a Pekin Duck Flock to Duck Hepatitis A Virus Genotype 3Semantic proximity Read ↗

Transcriptome Analysis Reveals New Insights of Novel Duck Reovirus (NDRV)-Infected Duck Adherent PBMCsSemantic proximity Read ↗

Comparative pathogenicity of duck hepatitis A virus genotype 3 in different duck breeds: Implications of the diagnosis and prevention of duck viral hepatitis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.