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Article

Deletion of bglC triggers a genetic compensation response by awakening the expression of alternative beta-glucosidase.

Biochimica et biophysica acta. Gene regulatory mechanisms - 1 Oct 2020

Deflandre Benoit, Thiébaut Noémie, Planckaert Sören, Jourdan Samuel, Anderssen Sinaeda, Hanikenne Marc, Devreese Bart, Francis Isolde, Rigali Sébastien

Abstract excerpt

In the plant pathogen Streptomyces scabies, the gene bglC encodes a GH1 family cellobiose beta-glucosidase that is both required for primary metabolism and for inducing virulence of the bacterium. Deletion of bglC (strain ΔbglC) surprisingly resulted in the augmentation of the global beta-glucosidase activity of S. scabies. This paradoxical phenotype is highly robust as it has been observed in all bglC deletion...

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Identifiers

PMID: 32758700
DOI: 10.1016/j.bbagrm.2020.194615

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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