Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

MicroRNA‐214 contributes to regulation of necroptosis via targeting ATF4 in diabetes‐associated periodontitis

15 May 2019

Abstract excerpt

Diabetes and periodontal diseases have a mutual promoting relationship that induces severe tissue damage and cell death. The potential roles of microRNAs (miRNAs) and the type of cell death involved in diabetes-associated periodontitis are obscure. The gingival tissues of patients were obtained and MC3T3-E1 cells were costimulated with high glucose and lipopolysaccharide (LPS). Osseous morphometric analysis was...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell death mechanisms and regulation
Share to Erythrocyte Function and Pathophysiology
Share to Inflammasome and immune disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31090954
DOI: 10.1002/jcb.28740

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

MicroRNA‐214 contributes to regulation of necroptosis via targeting ATF4 in diabetes‐associated periodontitisPMID 31090954Read ↗

PINK1 Mediated Mitophagy Attenuates Early Apoptosis of Gingival Epithelial Cells Induced by High GlucoseSemantic proximity Read ↗

Role of ferroptosis-related genes in periodontitis based on integrated bioinformatics analysisSemantic proximity Read ↗

Type 2 Diabetes Mellitus Aggravates Pathological Damage in Chronic Pulpitis: Association with AGEs/RAGE-MAPK Signaling Pathway ActivationSemantic proximity Read ↗

Macrophage efferocytosis controls tissue repair via mitochondrial dynamics in diabetic periodontitisSemantic proximity Read ↗

Identification of ferroptosis, necroptosis, and pyroptosis-associated genes in periodontitis-affected human periodontal tissue using integrated bioinformatic analysisSemantic proximity Read ↗

Programmed cell death in periodontitis: recent advances and future perspectivesSemantic proximity Read ↗

Mixed lineage kinase domain-like pseudokinase mediated necroptosis aggravates periodontitis progressionSemantic proximity Read ↗

Regulation of the unfolded protein response transducer IRE1α by SERPINH1 aggravates periodontitis with diabetes mellitus via prolonged ER stressSemantic proximity Read ↗

Identification of cross-talk pathways and ferroptosis-related genes in periodontitis and type 2 diabetes mellitus by bioinformatics analysis and experimental validationSemantic proximity Read ↗

Attenuated NIX in impaired mitophagy contributes to exacerbating cellular senescence in experimental periodontitis under hyperglycemic conditionsSemantic proximity Read ↗

SUMO1 modification of IGF-1R combining with SLUG inhibited osteogenic differentiation of PDLSCs stimulated by high glucoseSemantic proximity Read ↗

Genetic Analysis of Exacerbated Healing Responses after Periodontal Therapy due to Type 2 Diabetes: Clinical and in vivo StudiesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.