Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Bidirectional Role of β2-Adrenergic Receptor in Autoimmune Diseases

27 Nov 2018

Abstract excerpt

Disorder of the sympathetic nervous system (SNS) is closely related to the pathogenesis of various autoimmune diseases (ADs). Catecholamine triggered beta2-adrenergic receptor (β2-AR) signaling is important in creating a bidirectional response in the progression of ADs due to factors including diverse expression patterns, single nucleotide polymorphisms (SNPs), biased signals, and desensitization of β2-AR, as...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Asthma and respiratory diseases
Share to Neuropeptides and Animal Physiology
Share to Receptor Mechanisms and Signaling

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30538630
DOI: 10.3389/fphar.2018.01313

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bidirectional Role of β2-Adrenergic Receptor in Autoimmune DiseasesPMID 30538630Read ↗

The α2A-adrenergic receptor on Tfh cells amplifies allergic airway inflammation via Akt signalingSemantic proximity Read ↗

Norepinephrine Inhibits Th17 Cells via β2-Adrenergic Receptor (β2-AR) Signaling in a Mouse Model of Rheumatoid Arthritis.Semantic proximity Read ↗

TLR9-activated B cells improve their regulatory function by endogenously produced catecholaminesSemantic proximity Read ↗

Sympathetic neural signaling via the β2‐adrenergic receptor suppresses T‐cell receptor‐mediated human and mouse CD8<sup>+</sup> T‐cell effector functionSemantic proximity Read ↗

The role of adenosinergic pathway in human autoimmune diseasesSemantic proximity Read ↗

β2 adrenergic receptor–mediated signaling regulates the immunosuppressive potential of myeloid-derived suppressor cellsSemantic proximity Read ↗

β1-Adrenergic Receptor Function, Autoimmunity, and Pathogenesis of Dilated CardiomyopathySemantic proximity Read ↗

What Makes Antibodies Against G Protein-Coupled Receptors so Special? A Novel Concept to Understand Chronic DiseasesSemantic proximity Read ↗

β2-Adrenergic Biased Agonist Nebivolol Inhibits the Development of Th17 and the Response of Memory Th17 Cells in an NF-κB-Dependent MannerSemantic proximity Read ↗

Neuroendocrine‐Immune InteractionsSemantic proximity Read ↗

Autoimmune polyendocrine syndromes.Semantic proximity Read ↗

Pharmacology and Physiology of Human Adrenergic Receptor polymorphismsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.