Disturbance of the Epithelial-Mesenchymal Transition as a Cause of the Defective Development of Human Spontaneous Abortions with Abnormal Karyotype.

doi:10.1007/s10517-018-4217-4

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Disturbance of the Epithelial-Mesenchymal Transition as a Cause of the Defective Development of Human Spontaneous Abortions with Abnormal Karyotype.

Bulletin of experimental biology and medicine - 1 Aug 2018

Kukharenko V I

Abstract excerpt

Genetic program regulating epithelial-mesenchymal transition is activated at the early stages of embryogenesis. Abnormal karyotype of the embryo disrupts the fundamental process of epithelial-mesenchymal transition, which is seen from incomplete morphological rearrangement and reduced migration capacity of cells, as well as low level of expression of epithelial-mesenchymal transition markers (fibronectin,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Abnormal Karyotype
Share to Abortion, Spontaneous
Share to Collagen
Share to Epithelial-Mesenchymal Transition

Source

Open in PubMed

Identifiers

PMID: 30121915
DOI: 10.1007/s10517-018-4217-4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Fibronectins

Share to Glycosaminoglycans

Share to Nervous System Malformations

Share to Trisomy