Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Long-term Persistent Organic Pollutants Exposure Induced Telomere Dysfunction and Senescence-Associated Secretary Phenotype

19 Jan 2018

Abstract excerpt

Environmentally persistent organic pollutant (POP) is the general term for refractory organic compounds that show long-range atmospheric transport, environmental persistence, and bioaccumulation. It has been reported that the accumulation of POPs could lead to cellular DNA damage and adverse effects of on metabolic health. To better understand the mechanism of the health risks associated with POPs, we conducted...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Air Quality and Health Impacts
Share to Microplastics and Plastic Pollution
Share to Telomeres, Telomerase, and Senescence

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 29360938
DOI: 10.1093/gerona/gly002

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Long-term Persistent Organic Pollutants Exposure Induced Telomere Dysfunction and Senescence-Associated Secretary PhenotypePMID 29360938Read ↗

Environmental pollutants exposure: A potential contributor for aging and age-related diseasesSemantic proximity Read ↗

Revealing toxic secrets: How PAHs accelerate cellular ageing and trigger human diseasesSemantic proximity Read ↗

The Chemical Exposome of Human AgingSemantic proximity Read ↗

Republished: Environmental and occupational exposure to chemicals and telomere length in human studiesSemantic proximity Read ↗

Exposure to Persistent Organic Pollutants Predicts Telomere Length in Older Age: Results from the Helsinki Birth Cohort StudySemantic proximity Read ↗

Integrating Environmental Exposures, Epigenetic Programming and Nanotechnology for Chronic Disease Prevention in the One Health EraSemantic proximity Read ↗

Environmental and occupational exposure to chemicals and telomere length in human studiesSemantic proximity Read ↗

Hallmarks of environmental insults.Semantic proximity Read ↗

Changes in hormones, Leukocyte Telomere Length (LTL), and p16INK4a expression in SM-exposed individuals in favor of the cellular senescenceSemantic proximity Read ↗

Changes in the expression of genes involved in cell cycle regulation and the relative telomere length in the process of canceration induced by omethoate.Semantic proximity Read ↗

Epigenetic impact of endocrine-disrupting chemicals on atherosclerosisSemantic proximity Read ↗

Long Non-Coding RNAs: A Novel Paradigm for Toxicology.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.