Replicated association of Synaptotagmin (SYT1) with ADHD and its broader influence in externalizing behaviors.

doi:10.1016/j.euroneuro.2017.01.007

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Replicated association of Synaptotagmin (SYT1) with ADHD and its broader influence in externalizing behaviors.

European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology - 1 Mar 2017

Cupertino Renata Basso, Schuch Jaqueline Bohrer, Bandeira Cibele Edom, da Silva Bruna Santos, Rovaris Diego Luiz, Kappel Djenifer B, Contini Verônica, Salatino-Oliveira Angélica, Vitola Eduardo Schneider, Karam Rafael Gomes, Hutz Mara Helena, Rohde Luis Augusto, Grevet Eugenio Horacio, Bau Claiton Henrique Dotto, Mota Nina Roth

Abstract excerpt

Attention-Deficit/Hyperactivity Disorder (ADHD) is a common psychiatric disorder, affecting both children and adults. The Soluble N-ethylmaleimide sensitive factor Attachment REceptors (SNARE) complex has been implicated in ADHD pathophysiology since it is a key component of neurotransmitter release events and neurodevelopment processes, and SNPs in this complex have been associated with ADHD. Here we aim to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antisocial Personality Disorder

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Attention Deficit Disorder with Hyperactivity

Share to Case-Control Studies

Share to Conduct Disorder

Share to Gene Frequency

Share to Genetic Association Studies

Share to Mental Disorders

Share to Synaptotagmin I

Source

Open in PubMed

Identifiers

PMID: 28130000
DOI: 10.1016/j.euroneuro.2017.01.007

Genes

SYT1
SNAP25
VAMP2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Replicated association of Synaptotagmin (SYT1) with ADHD and its broader influence in externalizing behaviors.PMID 28130000Read ↗

Evaluation of common variants in 16 genes involved in the regulation of neurotransmitter release in ADHD.Semantic proximity Read ↗

Association between genetic polymorphisms of synaptophysin (SYP) gene and attention deficit hyperactivity disorder in Korean subjects.Semantic proximity Read ↗

Expanding the genotype and phenotype spectrum of SYT1-associated neurodevelopmental disorder.Semantic proximity Read ↗

Dances with black widow spiders: dysregulation of glutamate signalling enters centre stage in ADHD.Semantic proximity Read ↗

Pharmacogenetic Role of the Synaptosomal-Associated Protein 25 Gene in Clinical Response to Methylphenidate Among Turkish Pediatric ADHD CohortSemantic proximity Read ↗

The neurodevelopmental spectrum of synaptic vesicle cycling disorders.Semantic proximity Read ↗

Adaptive functions correlate with evoked neurotransmitter release in SYT1-associated neurodevelopmental disorderSemantic proximity Read ↗

CDH2 mutation affecting N-cadherin function causes attention-deficit hyperactivity disorder in humans and mice.Semantic proximity Read ↗

Expanding the genetic and phenotypic spectrum of Baker-Gordon syndrome: a new de novo SYT1 variant.Semantic proximity Read ↗

SYT1 Gene-Wide Analysis on White Matter Microstructure in Adulthood ADHDSemantic proximity Read ↗

NOS1 and SNAP25 polymorphisms are associated with Attention-Deficit/Hyperactivity Disorder symptoms in adults but not in children.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.