Expression of truncated bile salt-dependent lipase variant in pancreatic pre-neoplastic lesions.

doi:10.18632/oncotarget.11777

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Expression of truncated bile salt-dependent lipase variant in pancreatic pre-neoplastic lesions.

Oncotarget - 3 Jan 2017

Martinez Emmanuelle, Crenon Isabelle, Silvy Françoise, Del Grande Jean, Mougel Alice, Barea Dolores, Fina Frederic, Bernard Jean-Paul, Ouaissi Mehdi, Lombardo Dominique, Mas Eric

Abstract excerpt

Pancreatic adenocarcinoma (PDAC) is a dismal disease. The lack of specific symptoms still leads to a delay in diagnosis followed by death within months for most patients. Exon 11 of the bile salt-dependent lipase (BSDL) gene encoding variable number of tandem repeated (VNTR) sequences has been involved in pancreatic pathologies. We hypothesized that BSDL VNTR sequences may be mutated in PDAC. The amplification of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Line
Share to Gene Expression

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Immunohistochemistry

Share to Pancreatic Neoplasms

Share to Precancerous Conditions

Share to Proto-Oncogene Proteins p21(ras)

Share to RNA, Messenger

Share to Sterol Esterase

Source

Open in PubMed Open full text

Identifiers

PMID: 27602750
DOI: 10.18632/oncotarget.11777
PMCID: PMC5352176

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.