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Article

Sp7/Osterix Is Restricted to Bone-Forming Vertebrates where It Acts as a Dlx Co-factor in Osteoblast Specification

30 Apr 2016

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 27134141
DOI: 10.1016/j.devcel.2016.04.002

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Related research

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Sp7/Osterix Is Restricted to Bone-Forming Vertebrates where It Acts as a Dlx Co-factor in Osteoblast SpecificationPMID 27134141Read ↗

Sp7/osterix positively regulates dlx2b and bglap to affect tooth development and bone mineralization in zebrafish larvaeSemantic proximity Read ↗

The Novel Zinc Finger-Containing Transcription Factor Osterix Is Required for Osteoblast Differentiation and Bone FormationSemantic proximity Read ↗

Recent Advances of Osterix Transcription Factor in Osteoblast Differentiation and Bone FormationSemantic proximity Read ↗

The bone-related Zn finger transcription factor Osterix promotes proliferation of mesenchymal cellsSemantic proximity Read ↗

BMP-2-induced Osterix expression is mediated by Dlx5 but is independent of Runx2Semantic proximity Read ↗

Transcriptional Regulation of Jaw Osteoblasts: Development to PathologySemantic proximity Read ↗

Osterix and RUNX2 are Transcriptional Regulators of Sclerostin in Human BoneSemantic proximity Read ↗

DLX Genes in the Development and Maintenance of the Vertebrate Skeleton: Implications for Human PathologiesSemantic proximity Read ↗

Role of DLX Regulatory Proteins in Osteogenesis and ChondrogenesisSemantic proximity Read ↗

Semaphorins in bone development, homeostasis, and diseaseSemantic proximity Read ↗

SP7: from Bone Development to Skeletal Disease.Semantic proximity Read ↗

Genetic and molecular control of osterix in skeletal formation.Semantic proximity Read ↗

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