Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Overview of the Genetic Basis and Epigenetic Mechanisms that Contribute to FASD Pathobiology

20 Apr 2016

Abstract excerpt

Prenatal alcohol (ethanol) exposure (PAE) is the underlying cause for a variety of birth defects and neurodevelopmental deficits referred to as "Fetal Alcohol Spectrum Disorders (FASD)". The more visible phenotypes caused by PAE include growth retardation, and characteristic craniofacial abnormalities associated with functional and structural damage to the central nervous system. Ethanol is a teratogenic agent...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Metabolism and Genetic Disorders
Share to Prenatal Substance Exposure Effects

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 27086780
DOI: 10.2174/1568026616666160414124816

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 23 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Overview of the Genetic Basis and Epigenetic Mechanisms that Contribute to FASD PathobiologyPMID 27086780Read ↗

Fetal alcohol spectrum disorders: Genetic and epigenetic mechanismsSemantic proximity Read ↗

Genetic and epigenetic insights into fetal alcohol spectrum disordersSemantic proximity Read ↗

Review and gap analysis: molecular pathways leading to fetal alcohol spectrum disordersSemantic proximity Read ↗

Fetal alcohol spectrum disorders and their transmission through genetic and epigenetic mechanismsSemantic proximity Read ↗

Epigenetic Impacts of Early Life Stress in Fetal Alcohol Spectrum Disorders Shape the Neurodevelopmental ContinuumSemantic proximity Read ↗

Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: a model for fetal alcohol spectrum disordersSemantic proximity Read ↗

Fetal Alcohol Spectrum Disorder (FASD) Associated Neural Defects: Complex Mechanisms and Potential Therapeutic TargetsSemantic proximity Read ↗

Gene-ethanol interactions underlying fetal alcohol spectrum disorders.Semantic proximity Read ↗

The Impact of Oxidative Stress on the Epigenetics of Fetal Alcohol Spectrum DisordersSemantic proximity Read ↗

The Impact of Oxidative Stress on the Epigenetics of Fetal Alcohol Spectrum DisordersSemantic proximity Read ↗

Epigenetics Studies of Fetal Alcohol Spectrum Disorder: Where are We Now?Semantic proximity Read ↗

Genetic Influences on Fetal Alcohol Spectrum DisorderSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.