Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

IBR5 Modulates Temperature-Dependent, R Protein CHS3-Mediated Defense Responses in Arabidopsis.

PLoS genetics - 1 Oct 2015

Liu Jingyan, Yang Haibian, Bao Fei, Ao Kevin, Zhang Xiaoyan, Zhang Yuelin, Yang Shuhua

Abstract excerpt

Plant responses to low temperature are tightly associated with defense responses. We previously characterized the chilling-sensitive mutant chs3-1 resulting from the activation of the Toll and interleukin 1 receptor-nucleotide binding-leucine-rich repeat (TIR-NB-LRR)-type resistance (R) protein harboring a C-terminal LIM (Lin-11, Isl-1 and Mec-3 domains) domain. Here we report the identification of a suppressor...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arabidopsis Proteins
Share to Cell Cycle Proteins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chitin Synthase

Share to Cold Temperature

Share to Disease Resistance

Share to Dual-Specificity Phosphatases

Share to HSP90 Heat-Shock Proteins

Share to Plant Diseases

Source

Open in PubMed Open full text

Identifiers

PMID: 26451844
DOI: 10.1371/journal.pgen.1005584
PMCID: PMC4599859

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

IBR5 Modulates Temperature-Dependent, R Protein CHS3-Mediated Defense Responses in Arabidopsis.PMID 26451844Read ↗

A mutant CHS3 protein with TIR-NB-LRR-LIM domains modulates growth, cell death and freezing tolerance in a temperature-dependent manner in Arabidopsis.Semantic proximity Read ↗

A missense mutation in CHS1, a TIR-NB protein, induces chilling sensitivity in Arabidopsis.Semantic proximity Read ↗

Temperature-dependent autoimmunity mediated by chs1 requires its neighboring TNL gene SOC3.Semantic proximity Read ↗

Arabidopsis HSP90 protein modulates RPP4-mediated temperature-dependent cell death and defense responses.Semantic proximity Read ↗

Autoimmunity conferred by chs3-2D relies on CSA1, its adjacent TNL-encoding neighbour.Semantic proximity Read ↗

A gain-of-function mutation in the Arabidopsis disease resistance gene RPP4 confers sensitivity to low temperature.Semantic proximity Read ↗

A TIR-NBS protein encoded by Arabidopsis Chilling Sensitive 1 (CHS1) limits chloroplast damage and cell death at low temperature.Semantic proximity Read ↗

Genetic analyses of autoimmune mutants uncover the MPK3/6-CHR5-SNC1 module in Arabidopsis.Semantic proximity Read ↗

PBL13 Is a Serine/Threonine Protein Kinase That Negatively Regulates Arabidopsis Immune Responses.Semantic proximity Read ↗

Conserved role of the SERK–BIR module in development and immunity across land plantsSemantic proximity Read ↗

Regulation of transcription of nucleotide-binding leucine-rich repeat-encoding genes SNC1 and RPP4 via H3K4 trimethylation.Semantic proximity Read ↗

The HIRA-interacting histone chaperone CABIN1 negatively regulates plant immunity mediated by immune receptor gene SNC1.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.