Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Dissecting susceptibility from exogenous triggers: the model of alopecia areata and associated inflammatory skin diseases.

Journal of the European Academy of Dermatology and Venereology : JEADV - 1 Dec 2015

Garzorz N, Alsisi M, Todorova A, Atenhan A, Thomas J, Lauffer F, Ring J, Schmidt-Weber C, Biedermann T, Eyerich S, Eyerich K

Abstract excerpt

BACKGROUND: Alopecia areata (AA) is a T-cell-driven autoimmune disease of the hair follicle and frequently reported to be associated with inflammatory skin diseases (ISD) such as atopic eczema (AE) or psoriasis. Interestingly, AA on the one hand and both AE and psoriasis on the other hand are believed to be driven by mutually antagonistic T-cell subsets. OBJECTIVE: To characterize AA-specific T-cell profiles and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alopecia Areata
Share to CD4-CD8 Ratio

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to CD4-Positive T-Lymphocytes

Share to CD8-Positive T-Lymphocytes

Share to Dermatitis, Atopic

Share to Interferon-gamma

Share to Interleukin-17

Share to Psoriasis

Source

Identifiers

PMID: 26416203
DOI: 10.1111/jdv.13325

Genes

TNF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Dissecting susceptibility from exogenous triggers: the model of alopecia areata and associated inflammatory skin diseases.PMID 26416203Read ↗

The autoimmune basis of alopecia areata: a comprehensive review.Semantic proximity Read ↗

Deciphering the Complex Immunopathogenesis of Alopecia AreataSemantic proximity Read ↗

Alopecia areata: pathogenesis and potential for therapy.Semantic proximity Read ↗

So close, and yet so far away: The dichotomy of the specific immune response and inflammation in psoriasis and atopic dermatitis.Semantic proximity Read ↗

Alopecia areata: Disease characteristics, clinical evaluation, and new perspectives on pathogenesis.Semantic proximity Read ↗

Serum T helper 1 cytokine levels are greater in patients with alopecia areata regardless of severity or atopy.Semantic proximity Read ↗

Healthy individuals genetically at-risk for the development of Pemphigus vulgaris or Alopecia areata share disease-like cytokine dysregulation.Semantic proximity Read ↗

Genome-wide association study of atopic and autoimmune comorbidities in alopecia areata.Semantic proximity Read ↗

Pathophysiology of Alopecia Areata in the Pediatric PatientSemantic proximity Read ↗

Abnormal interactions between perifollicular mast cells and CD8+ T-cells may contribute to the pathogenesis of alopecia areata.Semantic proximity Read ↗

A transcriptomic map of murine and human alopecia areataSemantic proximity Read ↗

Clinical and Genetic Aspects of Alopecia Areata: A Cutting Edge Review.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.