Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Neurofilament light gene deletion exacerbates amyloid, dystrophic neurite, and synaptic pathology in the APP/PS1 transgenic model of Alzheimer's disease

10 Jul 2015

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alzheimer's disease research and treatments
Share to Prion Diseases and Protein Misfolding
Share to Skin and Cellular Biology Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26344875
DOI: 10.1016/j.neurobiolaging.2015.07.003

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Neurofilament light gene deletion exacerbates amyloid, dystrophic neurite, and synaptic pathology in the APP/PS1 transgenic model of Alzheimer's diseasePMID 26344875Read ↗

Early onset amyloid lesions lead to severe neuritic abnormalities and local, but not global neuron loss in APPPS1 transgenic miceSemantic proximity Read ↗

Loss of tau elicits axonal degeneration in a mouse model of Alzheimer's diseaseSemantic proximity Read ↗

Intraneuronal Aβ accumulation and neurodegeneration: Lessons from transgenic modelsSemantic proximity Read ↗

Amyloid β accumulation and inner retinal degenerative changes in Alzheimer’s disease transgenic mouseSemantic proximity Read ↗

Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formationSemantic proximity Read ↗

Hippocampal Neuron Loss Exceeds Amyloid Plaque Load in a Transgenic Mouse Model of Alzheimer's DiseaseSemantic proximity Read ↗

Neuroanatomical Abnormalities in Behaviorally Characterized APPV717F Transgenic MiceSemantic proximity Read ↗

Characterization of amyloid deposition in the APPswe/PS1dE9 mouse model of Alzheimer diseaseSemantic proximity Read ↗

Comparison of memory, affective behavior, and neuropathology in APPNLGF knock-in mice to 5xFAD and APP/PS1 miceSemantic proximity Read ↗

BACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic miceSemantic proximity Read ↗

Characterization of neuronal dystrophy induced by fibrillar amyloid β: implications for Alzheimer’s diseaseSemantic proximity Read ↗

Neuron loss in APP transgenic miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.