Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Restoration of miR-424 suppresses BCR–ABL activity and sensitizes CML cells to imatinib treatment

16 Feb 2015

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chronic Myeloid Leukemia Treatments
Share to Eosinophilic Disorders and Syndromes
Share to T-cell and Retrovirus Studies

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 25697481
DOI: 10.1016/j.canlet.2015.02.031

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Restoration of miR-424 suppresses BCR–ABL activity and sensitizes CML cells to imatinib treatmentPMID 25697481Read ↗

miR-29b suppresses CML cell proliferation and induces apoptosis via regulation of BCR/ABL1 proteinSemantic proximity Read ↗

MiR-30e induces apoptosis and sensitizes K562 cells to imatinib treatment via regulation of the BCR–ABL proteinSemantic proximity Read ↗

Genetic and Epigenetic Silencing of MicroRNA-203 Enhances ABL1 and BCR-ABL1 Oncogene ExpressionSemantic proximity Read ↗

Genetic and Epigenetic Silencing of MicroRNA-203 Enhances ABL1 and BCR-ABL1 Oncogene ExpressionSemantic proximity Read ↗

Myc induced miR-144/451 contributes to the acquired imatinib resistance in chronic myelogenous leukemia cell K562Semantic proximity Read ↗

Stable RNA interference (RNAi) as an option for anti-bcr-abl therapySemantic proximity Read ↗

HH-GV-678, a novel selective inhibitor of Bcr-Abl, outperforms imatinib and effectively overrides imatinib resistanceSemantic proximity Read ↗

LIN28A-Dependent Kinome and Phosphoproteome Reprogramming Promotes Imatinib ResistanceSemantic proximity Read ↗

Expression differences of miR-142-5p between treatment-naïve chronic myeloid leukemia patients responding and non-responding to imatinib therapy suggest a link to oncogenic ABL2, SRI, cKIT and MCL1 signaling pathways critical for development of therapy resistanceSemantic proximity Read ↗

MicroRNA-212/ABCG2-axis contributes to development of imatinib-resistance in leukemic cellsSemantic proximity Read ↗

PP2A inhibition sensitizes cancer stem cells to ABL tyrosine kinase inhibitors in BCR-ABL + human leukemiaSemantic proximity Read ↗

SHP2 is required for BCR-ABL1-induced hematologic neoplasiaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.