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Article

A Critical Role for Transcription Factor Smad4 in T Cell Function that Is Independent of Transforming Growth Factor β Receptor Signaling

25 Dec 2014

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 25577439
DOI: 10.1016/j.immuni.2014.12.019

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Related research

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A Critical Role for Transcription Factor Smad4 in T Cell Function that Is Independent of Transforming Growth Factor β Receptor SignalingPMID 25577439Read ↗

The multifaceted role of SMAD4 in immune cell functionSemantic proximity Read ↗

Smad4 signalling in T cells is required for suppression of gastrointestinal cancerSemantic proximity Read ↗

Consequences of Mutations and Abnormal Expression of SMAD4 in Tumors and T CellsSemantic proximity Read ↗

The tumor suppressor Smad4/DPC 4 as a central mediator of Smad functionSemantic proximity Read ↗

SMAD4 TGF-β–independent function preconditions naive CD8+ T cells to prevent severe chronic intestinal inflammationSemantic proximity Read ↗

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SMADs: mediators and regulators of TGF-β signalingSemantic proximity Read ↗

Smad proteins and transforming growth factor-β signalingSemantic proximity Read ↗

Partnership between DPC4 and SMAD proteins in TGF-β signalling pathwaysSemantic proximity Read ↗

The Transcription Factor NFAT4 Is Involved in the Generation and Survival of T CellsSemantic proximity Read ↗

TGF-β signalling through the Smad pathwaySemantic proximity Read ↗

Nucleocytoplasmic Shuttling of Smads 2, 3, and 4 Permits Sensing of TGF-β Receptor ActivitySemantic proximity Read ↗

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