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Article

A 'silent', new polymorphism of factor H and apparent de novo atypical haemolytic uraemic syndrome after kidney transplantation.

BMJ case reports - 23 Dec 2014

Broeders Emine N, Stordeur Patrick, Rorive Sandrine, Dahan Karin

Abstract excerpt

The pathophysiology of atypical haemolytic-uraemic syndrome (aHUS) occurring de novo after renal transplantation may include genetic mutations of regulators of complement activation, but they are still rarely determined. A 41-year-old female renal transplant recipient presented two very different episodes of thrombotic microangiopathy. The first episode was associated with antibody-mediated rejection and the...

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Share to Antibodies, Monoclonal, Humanized
Share to Atypical Hemolytic Uremic Syndrome

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Complement Factor H

Share to Graft Rejection

Share to Kidney

Share to Kidney Transplantation

Share to Polymorphism, Single Nucleotide

Share to Postoperative Complications

Source

Open in PubMed Open full text

Identifiers

PMID: 25538218
DOI: 10.1136/bcr-2014-207630
PMCID: PMC4275717

Genes

CFH

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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