Association of dopamine gene variants, emotion dysregulation and ADHD in autism spectrum disorder.

doi:10.1016/j.ridd.2014.04.007

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Association of dopamine gene variants, emotion dysregulation and ADHD in autism spectrum disorder.

Research in developmental disabilities - 1 Jul 2014

Gadow Kenneth D, Pinsonneault Julia K, Perlman Greg, Sadee Wolfgang

Abstract excerpt

The aim of the present study was to evaluate the association of dopaminergic gene variants with emotion dysregulation (EMD) and attention-deficit/hyperactivity disorder (ADHD) symptoms in children with autism spectrum disorder (ASD). Three dopamine transporter gene (SLC6A3/DAT1) polymorphisms (intron8 5/6 VNTR, 3'-UTR 9/10 VNTR, rs27072 in the 3'-UTR) and one dopamine D2 receptor gene (DRD2) variant (rs2283265)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Affective Symptoms

Share to Anxiety Disorders

Share to Attention Deficit Disorder with Hyperactivity

Share to Child Development Disorders, Pervasive

Share to Comorbidity

Share to Depressive Disorder, Major

Share to Dopamine Plasma Membrane Transport Proteins

Share to Receptors, Dopamine D2

Source

Open in PubMed Open full text

Identifiers

PMID: 24780147
DOI: 10.1016/j.ridd.2014.04.007
PMCID: PMC4084560

Genes

DRD2
EMD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of dopamine gene variants, emotion dysregulation and ADHD in autism spectrum disorder.PMID 24780147Read ↗

Association of ADHD, tics, and anxiety with dopamine transporter (DAT1) genotype in autism spectrum disorder.Semantic proximity Read ↗

Analysis of neurotransmitters validates the importance of the dopaminergic system in autism spectrum disorder.Semantic proximity Read ↗

Association between autism spectrum disorder and polymorphisms in genes encoding serotine and dopamine receptors.Semantic proximity Read ↗

Association of DRD4 polymorphism with severity of oppositional defiant disorder, separation anxiety disorder and repetitive behaviors in children with autism spectrum disorder.Semantic proximity Read ↗

Association of COMT (Val158Met) and BDNF (Val66Met) gene polymorphisms with anxiety, ADHD and tics in children with autism spectrum disorder.Semantic proximity Read ↗

Association of a monoamine oxidase-a gene promoter polymorphism with ADHD and anxiety in boys with autism spectrum disorder.Semantic proximity Read ↗

Emotion dysregulation in ADHD and other neurodevelopmental conditions: a co-twin control studySemantic proximity Read ↗

The dopamine hypothesis for ADHD: An evaluation of evidence accumulated from human studies and animal modelsSemantic proximity Read ↗

Toward operationalizing deficient emotional self-regulation in newly referred adults with ADHD: A receiver operator characteristic curve analysisSemantic proximity Read ↗

The role of COMT and plasma proline in the variable penetrance of autistic spectrum symptoms in 22q11.2 deletion syndrome.Semantic proximity Read ↗

Associations of endocrine stress-related gene polymorphisms with risk of autism spectrum disorders: Evidence from an integrated meta-analysis.Semantic proximity Read ↗

DRD2 and PPP1R1B (DARPP-32) polymorphisms independently confer increased risk for autism spectrum disorders and additively predict affected status in male-only affected sib-pair families.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.