Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Emerging genetic basis of osteochondritis dissecans.

Clinics in sports medicine - 1 Apr 2014

Bates J Tyler, Jacobs John C, Shea Kevin G, Oxford Julia Thom

Abstract excerpt

Genome-wide association studies (GWAS) provide an unbiased approach in the identification of genes that increase the risk for osteochondritis dissecans (OCD). Recent GWAS in humans, horses, and pigs are reviewed and genes identified. The identified genes tended to cluster with respect to function and biologic processes. GWAS in humans are a critical next step in the effort to provide a better understanding of the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Extracellular Matrix
Share to Genetic Predisposition to Disease
Share to Genotype

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Horse Diseases

Share to Horses

Share to Osteoarthritis Treatment and Mechanisms

Share to Swine Diseases

Source

Open in PubMed Open full text

Identifiers

PMID: 24698039
DOI: 10.1016/j.csm.2013.11.004
PMCID: PMC3976886

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Emerging genetic basis of osteochondritis dissecans.PMID 24698039Read ↗

A genome-wide association study of osteochondritis dissecans in the Thoroughbred.Semantic proximity Read ↗

Candidate Loci are Revealed by an Initial Genome-wide Association Study of Juvenile Osteochondritis Dissecans.Semantic proximity Read ↗

Osteochondrosis in horses: a review of non-genetic and genetic factors associated.Semantic proximity Read ↗

Osteochondrosis in horses: An overview of genetic and other factorsSemantic proximity Read ↗

Familial osteochondritis dissecans associated with early osteoarthritis and disproportionate short stature.Semantic proximity Read ↗

Genome-wide association analysis of osteochondrosis of the tibiotarsal joint in Norwegian Standardbred trotters.Semantic proximity Read ↗

Osteochondritis Dissecans Lesions in Family Members: Does a Positive Family History Impact Phenotypic Potency?Semantic proximity Read ↗

Gene expression profiling from leukocytes of horses affected by osteochondrosisSemantic proximity Read ↗

An across-breed validation study of 46 genetic markers in canine hip dysplasia.Semantic proximity Read ↗

Associations between candidate gene markers at a quantitative trait locus on equine chromosome 4 responsible for osteochondrosis dissecans in fetlock joints of South German Coldblood horses.Semantic proximity Read ↗

Genetic dissection of canine hip dysplasia phenotypes and osteoarthritis reveals three novel loci.Semantic proximity Read ↗

A missense mutation in the aggrecan C-type lectin domain disrupts extracellular matrix interactions and causes dominant familial osteochondritis dissecans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.