Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

[Relation of -55CT polymorphism of UCP3 gene with weight loss and metabolic changes after a high polyunsaturated fat diet in obese patients].

Nutricion hospitalaria - 1 Jan 2000

de Luis D A, Aller R, Izaola O, GonzÁlez Sagrado M, Conde R, Ruiz Mambrilla M

Abstract excerpt

BACKGROUND AND OBJECTIVES: The alteration in the protein expression of UCP3 could reduce energy consumption and increase energy storage as fat. The aim of our study was to investigate the influence of -55CT polymorphism of UCP3 gene in the metabolic response, weight loss and serum levels of adipokines following a hypocaloric diet rich in polyunsaturated fat in obese patients. DESIGN: A sample of 133 obese...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adipokines
Share to Body Mass Index

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fatty Acids, Unsaturated

Share to Ion Channels

Share to Mitochondrial Proteins

Share to Obesity

Share to Uncoupling Protein 3

Share to Weight Loss

Source

Identifiers

PMID: 23165529
DOI: 10.3305/nh.2012.27.4.5787

Genes

UCP3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

[Relation of -55CT polymorphism of UCP3 gene with weight loss and metabolic changes after a high polyunsaturated fat diet in obese patients].PMID 23165529Read ↗

Modulation of adipocytokines response and weight loss secondary to a hypocaloric diet in obese patients by -55CT polymorphism of UCP3 gene.Semantic proximity Read ↗

Modulation of insulin concentrations and metabolic parameters in obese patients by -55CT polymorphism of the UCP3 gene secondary to two hypocaloric diets.Semantic proximity Read ↗

Effect of -55CT Polymorphism of UCP3 on Insulin Resistance and Cardiovascular Risk Factors after a High Protein/Low Carbohydrate versus a Standard Hypocaloric Diet.Semantic proximity Read ↗

Association of -55CT polymorphism of UCP3 gene with fat distribution, cardiovascular risk factors and adipocytokines in patients with Type 2 diabetes mellitus.Semantic proximity Read ↗

Lack of association of -55CT polymorphism of UCP3 gene with fat distribution in obese patients.Semantic proximity Read ↗

Effects of genetic polymorphisms of UCP2 and UCP3 on very low calorie diet-induced body fat reduction in Korean female subjects.Semantic proximity Read ↗

The energy intake modulates the association of the -55CT polymorphism of UCP3 with body weight in type 2 diabetic patients.Semantic proximity Read ↗

[Effects of uncoupling protein 3 gene -55 C-->T variant on lipid metabolism, body fat, its distribution and non-insulin-dependent diabetes mellitus in Chinese].Semantic proximity Read ↗

Association of UCP3 Gene –55C>T Polymorphism and Obesity in a Spanish PopulationSemantic proximity Read ↗

Fatty acid-binding protein 2 Ala54Thr genotype is associated with insulin resistance and leptin levels changes after a high monounsaturated fat diet in obese non-diabetic patients.Semantic proximity Read ↗

The common -55 C/T polymorphism in the promoter region of the uncoupling protein 3 gene reduces prevalence of obesity and elevates serum high-density lipoprotein cholesterol levels in the general Japanese population.Semantic proximity Read ↗

Relationship of -55C/T polymorphism of uncoupling protein 3 (UCP3) gene with metabolic syndrome by ATP III classification.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.