Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

n-Butyl Benzyl Phthalate Promotes Breast Cancer Progression by Inducing Expression of Lymphoid Enhancer Factor 1

8 Aug 2012

Abstract excerpt

Environmental hormones play important roles in regulating the expression of genes involved in cell proliferation, drug resistance, and breast cancer risk; however, their precise role in human breast cancer cells during cancer progression remains unclear. To elucidate the effect of the most widely used industrial phthalate, n-butyl benzyl phthalate (BBP), on cancer progression, we evaluated the results of BBP...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Breast Cancer Treatment Studies
Share to Cancer Cells and Metastasis
Share to Estrogen and related hormone effects

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 22905168
DOI: 10.1371/journal.pone.0042750

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

n-Butyl Benzyl Phthalate Promotes Breast Cancer Progression by Inducing Expression of Lymphoid Enhancer Factor 1PMID 22905168Read ↗

The chemical environmental pollutants BPA and BPS induce alterations of the proteomic profile of different phenotypes of human breast cancer cells: A proposed interactome.Semantic proximity Read ↗

Parabens Promote Pro-Tumorigenic Effects in Luminal Breast Cancer Cell Lines with Diverse Genetic AncestrySemantic proximity Read ↗

Reduction of daily-use parabens and phthalates reverses accumulation of cancer-associated phenotypes within disease-free breast tissue of study subjects.Semantic proximity Read ↗

Breast Cancer ExposomicsSemantic proximity Read ↗

Toxicity and mechanistic analysis of Di(2- ethylhexyl)phthalate in renal cell carcinoma Progression: a systematic study with network toxicology and molecular docking strategiesSemantic proximity Read ↗

Exposure to the phthalate metabolite MEHP impacts survival and growth of human ovarian follicles in vitroSemantic proximity Read ↗

Exposure to Phthalate, an Endocrine Disrupting Chemical, Alters the First Trimester Placental Methylome and Transcriptome in WomenSemantic proximity Read ↗

Urinary concentration of endocrine-disrupting phthalates and breast cancer risk in Indian women: A case-control study with a focus on mutations in phthalate-responsive genes.Semantic proximity Read ↗

Network Toxicology and Molecular Docking Reveal Diethyl Phthalate as a Potential Epigenetic Modulator in Psoriasis PathogenesisSemantic proximity Read ↗

Long-term dietary exposure to a mixture of phthalates enhances estrogen and beta-catenin signaling pathways, leading to endometrial hyperplasia in miceSemantic proximity Read ↗

Mono-(2-Ethylhexyl) Phthalate (MEHP) Promotes Invasion and Migration of Human Testicular Embryonal Carcinoma Cells1Semantic proximity Read ↗

Dibutyl phthalate disrupts conserved circadian rhythm in Drosophila and human cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.