Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

No association of SIDS with two polymorphisms in genes relevant for the noradrenergic system: COMT and DBH.

Acta paediatrica (Oslo, Norway : 1992) - 1 Oct 2012

Klintschar Michael, Heimbold Christian

Abstract excerpt

AIM: Recent research suggests that genetic variance determining the strength of noradrenergic transmitting might contribute to the aetiology of SIDS. We have typed 2 functional polymorphisms of relevance for both biosynthesis and catabolism of noradrenalin: The Val158Met single-nucleotide polymor...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Catechol O-Methyltransferase
Share to Chi-Square Distribution
Share to Dopamine

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Germany

Share to Infant

Share to Infant, Newborn

Share to Sudden Infant Death

Share to Synaptic Transmission

Source

Identifiers

PMID: 22765285
DOI: 10.1111/j.1651-2227.2012.02764.x

Genes

COMT
DBH

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-22765285-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

No association of SIDS with two polymorphisms in genes relevant for the noradrenergic system: COMT and DBH.PMID 22765285Read ↗

Gene variants predisposing to SIDS: current knowledge.Semantic proximity Read ↗

A functional polymorphism in the tyrosine hydroxylase gene indicates a role of noradrenalinergic signaling in sudden infant death syndrome.Semantic proximity Read ↗

Genetic Association Study of Acetylcholinesterase (ACHE) and Butyrylcholinesterase (BCHE) Variants in Sudden Infant Death Syndrome (SIDS).Semantic proximity Read ↗

Sudden infant death syndrome (SIDS) and polymorphisms in Monoamine oxidase A gene (MAOA): a revisit.Semantic proximity Read ↗

Association between a functional polymorphism in the MAOA gene and sudden infant death syndrome.Semantic proximity Read ↗

Tyrosine hydroxylase TH01 9.3 allele in the occurrence of sudden infant death syndrome in Swiss Caucasians.Semantic proximity Read ↗

Significant association of TH01 allele 9.3 and SIDS.Semantic proximity Read ↗

Gene variants associated with obstructive sleep apnea (OSA) in relation to sudden infant death syndrome (SIDS).Semantic proximity Read ↗

That's not it, either-neither polymorphisms in PHOX2B nor in MIF are involved in sudden infant death syndrome (SIDS).Semantic proximity Read ↗

Association between monoamine oxidase A promoter polymorphism and the risk of sudden infant death syndrome: a meta-analysis.Semantic proximity Read ↗

HTR2A variation and sudden infant death syndrome: a case-control analysis.Semantic proximity Read ↗

Genetic variation in the monoamine oxidase A and serotonin transporter genes in sudden infant death syndrome.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.