Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Relationship among metabolic syndrome, C358A polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) and insulin resistance.

Journal of diabetes and its complications - 1 Jan 2000

de Luis D A, Aller R, Izaola O, Conde R, Sagrado M Gonzalez, Primo D, Castro M J

Abstract excerpt

BACKGROUND: It has been demonstrated that the polymorphism 385 C/A of FAAH (fatty acid amide hydrolase) was associated with obesity and metabolic disorders. OBJECTIVE: The aim of our study was to investigate the relationship of the polymorphism (cDNA 385 C->A) of FAAH gene and insulin resistance in obese patients with and without metabolic syndrome. DESIGN: A population of 799 obese patients was analyzed in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amidohydrolases
Share to Comorbidity

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cross-Sectional Studies

Share to Homeostasis

Share to Insulin

Share to Insulin Resistance

Share to Metabolic Syndrome

Share to Obesity

Source

Identifiers

PMID: 22609216
DOI: 10.1016/j.jdiacomp.2012.04.002

Genes

FAAH

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Relationship among metabolic syndrome, C358A polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) and insulin resistance.PMID 22609216Read ↗

Relation of C358A polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) with obesity and insulin resistance.Semantic proximity Read ↗

C358A missense polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) and insulin resistance in patients with diabetes mellitus type 2.Semantic proximity Read ↗

[C358A polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) and adipocytokine levels in the morbidly obese].Semantic proximity Read ↗

Genetic variation in the endocannabinoid degrading enzyme fatty acid amide hydrolase (FAAH) and their influence on weight loss and insulin resistance under a high monounsaturated fat hypocaloric diet.Semantic proximity Read ↗

C358A missense polymorphism of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (FAAH) and visfatin levels in obese females.Semantic proximity Read ↗

Association between the FAAH C385A variant (rs324420) and obesity-related traits: a systematic review.Semantic proximity Read ↗

Fatty acid amide hydrolase C385A polymorphism affects susceptibility to various diseases.Semantic proximity Read ↗

Circulating Endocannabinoids and the Polymorphism 385C>A in Fatty Acid Amide Hydrolase (FAAH) Gene May Identify the Obesity Phenotype Related to Cardiometabolic Risk: A Study Conducted in a Brazilian Population of Complex Interethnic Admixture.Semantic proximity Read ↗

Obesity-related dyslipidemia associated with FAAH, independent of insulin response, in multigenerational families of Northern European descent.Semantic proximity Read ↗

Racial and sex differences in the polymorphisms of the endocannabinoid receptor genes in obesity.Semantic proximity Read ↗

Genetic polymorphisms of the endocannabinoid system in obesity and diabetes.Semantic proximity Read ↗

Cholesteryl ester transfer protein (CETP) -629C/A polymorphism and it's effects on the serum lipid levels in metabolic syndrome patients.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.