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Article

Overexpression of miR-429 induces mesenchymal-to-epithelial transition (MET) in metastatic ovarian cancer cells

29 Jan 2011

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 21277012
DOI: 10.1016/j.ygyno.2010.12.339

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Related research

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Overexpression of miR-429 induces mesenchymal-to-epithelial transition (MET) in metastatic ovarian cancer cellsPMID 21277012Read ↗

Sequence variation among members of the miR-200 microRNA family is correlated with variation in the ability to induce hallmarks of mesenchymal-epithelial transition in ovarian cancer cells.Semantic proximity Read ↗

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miR-30d Blocked Transforming Growth Factor β1–Induced Epithelial-Mesenchymal Transition by Targeting Snail in Ovarian Cancer CellsSemantic proximity Read ↗

Mechanism of the mesenchymal-epithelial transition and its relationship with metastatic tumor formation.Semantic proximity Read ↗

MiR ‐429 suppresses proliferation and invasion of breast cancer via inhibiting the Wnt/β ‐catenin signaling pathwaySemantic proximity Read ↗

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<scp>MiR</scp>‐506 inhibits multiple targets in the epithelial‐to‐mesenchymal transition network and is associated with good prognosis in epithelial ovarian cancerSemantic proximity Read ↗

MiR-93-5p inhibits the EMT of breast cancer cells via targeting MKL-1 and STAT3Semantic proximity Read ↗

MiR-335 inhibits migration of breast cancer cells through targeting oncoprotein c-MetSemantic proximity Read ↗

miR-448 negatively regulates ovarian cancer cell growth and metastasis by targeting CXCL12Semantic proximity Read ↗

miR-1199-5p and Zeb1 function in a double-negative feedback loop potentially coordinating EMT and tumour metastasis.Semantic proximity Read ↗

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