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Article

Extracellular Hsp90 serves as a co-factor for MAPK activation and latent viral gene expression during de novo infection by KSHV

7 May 2010

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Share to Cytomegalovirus and herpesvirus research
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20451233
DOI: 10.1016/j.virol.2010.03.052

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Related research

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Research trail→

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Extracellular Hsp90 serves as a co-factor for MAPK activation and latent viral gene expression during de novo infection by KSHVPMID 20451233Read ↗

Cell signaling pathways engaged by KSHVSemantic proximity Read ↗

Early and sustained expression of latent and host modulating genes in coordinated transcriptional program of KSHV productive primary infection of human primary endothelial cellsSemantic proximity Read ↗

Broad action of Hsp90 as a host chaperone required for viral replicationSemantic proximity Read ↗

Reactivation and Lytic Replication of Kaposi’s Sarcoma-Associated Herpesvirus: An UpdateSemantic proximity Read ↗

Human herpesvirus KSHV encodes a constitutively active G-protein-coupled receptor linked to cell proliferationSemantic proximity Read ↗

Screening of the Human Kinome Identifies MSK1/2-CREB1 as an Essential Pathway Mediating Kaposi's Sarcoma-Associated Herpesvirus Lytic Replication during Primary InfectionSemantic proximity Read ↗

BiP/GRP78 is a pro-viral factor for diverse dsDNA viruses that promotes the survival and proliferation of cells upon KSHV infectionSemantic proximity Read ↗

Transactivation of human endogenous retrovirus K (HERV-K) by KSHV promotes Kaposi’s sarcoma developmentSemantic proximity Read ↗

Regulation of NF-κB inhibitor IκBα and viral replication by a KSHV microRNASemantic proximity Read ↗

Hepatitis C virus RNA replication is regulated by FKBP8 and Hsp90Semantic proximity Read ↗

KSHV-encoded ORF45 activates human NLRP1 inflammasomeSemantic proximity Read ↗

Biphasic Euchromatin-to-Heterochromatin Transition on the KSHV Genome Following De Novo InfectionSemantic proximity Read ↗

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