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Article

Is an endothelial nitric oxide synthase gene mutation a risk factor in the origin of intraventricular hemorrhage?

Neurosurgical focus - 1 Jan 2010

Vannemreddy Prasad, Notarianni Christina, Yanamandra Krishna, Napper Dawn, Bocchini Joseph

Abstract excerpt

OBJECT: Studies have shown decreased levels of nitric oxide (NO), the product of endothelial NO synthase (eNOS) gene activity, in infants with respiratory conditions and intraventricular hemorrhage (IVH). The authors evaluated the association of the eNOS gene promoter polymorphism T-786C with the...

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Share to Bronchopulmonary Dysplasia
Share to Cerebral Hemorrhage
Share to Cerebral Ventricles

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chromosomes, Human, Pair 7

Share to Infant, Premature

Share to Nitric Oxide

Share to Nitric Oxide Synthase Type III

Share to Respiratory Distress Syndrome, Newborn

Source

Identifiers

PMID: 20043715
DOI: 10.3171/2009.10.FOCUS09143

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20043715-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

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