Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Tumor marker phenotype concordance in second primary breast cancer, California, 1999-2004.

Breast cancer research and treatment - 1 Feb 2010

Brown Monica, Bauer Katrina, Pare Mary

Abstract excerpt

Breast cancer is the most common cancer among women. It is estimated that 7% of women who have breast cancer will develop a subsequent second independent breast tumor within 10 years of the first. The status of estrogen (ER), progesterone (PR) and human growth hormone (HER2) receptors, individual...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biomarkers, Tumor
Share to California
Share to Neoplasm Staging

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Neoplasms, Second Primary

Share to Receptor, ErbB-2

Share to Receptors, Estrogen

Share to Receptors, Progesterone

Share to Registries

Source

Identifiers

PMID: 19629680
DOI: 10.1007/s10549-009-0469-z

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-19629680-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Tumor marker phenotype concordance in second primary breast cancer, California, 1999-2004.PMID 19629680Read ↗

Molecular Concordance Between Primary Breast Cancer and Matched MetastasesSemantic proximity Read ↗

Tumor phenotype and concordance in synchronous bilateral breast cancer in young women.Semantic proximity Read ↗

Molecular Profiling of a Triple-negative Breast Cancer Cohort in India for EGFR and AR expression analyzed for patient outcomes showed a distinct subset of cellular co-expressionSemantic proximity Read ↗

Descriptive analysis of estrogen receptor (ER)-negative, progesterone receptor (PR)-negative, and HER2-negative invasive breast cancer, the so-called triple-negative phenotype: a population-based study from the California cancer Registry.Semantic proximity Read ↗

Receptor discordance and phenotype change in metastatic breast cancer.Semantic proximity Read ↗

Tumor phenotype and breast density in distinct categories of interval cancer: results of population-based mammography screening in SpainSemantic proximity Read ↗

Hormone receptor status of a first primary breast cancer predicts contralateral breast cancer risk in the WECARE study population.Semantic proximity Read ↗

Are bilateral breast cancers and breast cancers coexisting with ovarian cancer different from solitary tumors? A pair-matched immunohistochemical analysis aimed at intrinsic tumor phenotype.Semantic proximity Read ↗

Differential Second Primary Malignancy Occurrence After Breast Cancer According to HER2 Status: a SEER Population-Based AnalysisSemantic proximity Read ↗

Are synchronous and metachronous bilateral breast cancers different? An immunohistochemical analysis aimed at intrinsic tumor phenotype.Semantic proximity Read ↗

Synchronous Breast Cancer: Phenotypic Similarities on MRI.Semantic proximity Read ↗

Molecular phenotypes of DCIS predict overall and invasive recurrence.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.