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Article

RECQL4-deficient cells are hypersensitive to oxidative stress/damage: Insights for osteosarcoma prevalence and heterogeneity in Rothmund-Thomson syndrome

3 May 2006

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 16678792
DOI: 10.1016/j.bbrc.2006.04.093

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RECQL4-deficient cells are hypersensitive to oxidative stress/damage: Insights for osteosarcoma prevalence and heterogeneity in Rothmund-Thomson syndromePMID 16678792Read ↗

Rothmund–Thomson Syndrome Responsible Gene, RECQL4: Genomic Structure and ProductsSemantic proximity Read ↗

Rothmund-Thomson Syndrome: Insights from New Patients on the Genetic Variability Underpinning Clinical Presentation and Cancer Outcome.Semantic proximity Read ↗

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RECQL4-related Rothmund-Thomson syndrome: A case series and literature review.Semantic proximity Read ↗

Aging in Rothmund-Thomson syndrome and related RECQL4 genetic disorders.Semantic proximity Read ↗

The RECQL4 protein, deficient in Rothmund–Thomson syndrome is active on telomeric D-loops containing DNA metabolism blocking lesionsSemantic proximity Read ↗

RECQL4 in genomic instability and agingSemantic proximity Read ↗

Cancer risk among RECQL4 heterozygotes.Semantic proximity Read ↗

Functional characterisation of a RECQL4 mutation in Rothmund-Thomson syndromeSemantic proximity Read ↗

The versatile RECQL4.Semantic proximity Read ↗

Rare presentation of Rothmund-Thomson syndrome with novel compound heterozygous mutations of the RECQL4 gene.Semantic proximity Read ↗

Association between osteosarcoma and deleterious mutations in the RECQL4 gene in Rothmund-Thomson syndrome.Semantic proximity Read ↗

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