Biochemical models of hereditary pancreatitis.

doi:10.1016/j.ecl.2006.02.002

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Article

Biochemical models of hereditary pancreatitis.

Endocrinology and metabolism clinics of North America - 1 Jun 2006

Sahin-Tóth Miklós

Abstract excerpt

The past decade has witnessed remarkable progress in the genetics of chronic pancreatitis. Despite these accomplishments, the understanding of the molecular mechanisms through which PRSS1 and SPINK1 mutations cause chronic pancreatitis has remained sketchy. Pancreatitis-associated gene mutations are believed to result in uncontrolled trypsin activity in the pancreas. Experimental identification of the...

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Topics

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Share to Carrier Proteins
Share to Chronic Disease
Share to Genetic Variation
Share to Pancreatitis
Share to Point Mutation
Share to Trypsin
Share to Trypsin Inhibitor, Kazal Pancreatic
Share to Trypsinogen

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Identifiers

PMID: 16632094
DOI: 10.1016/j.ecl.2006.02.002
PMCID: PMC1602208

Genes

PRSS1
SPINK1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

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