Article
Preeclampsia: the Death of Goliath
19 Jan 2006
Abstract excerpt
In this review, a novel and unifying pathophysiologic mechanism of preeclampsia is presented whereby a minimal excess of placental immune complex production versus removal causes a proinflammatory autoamplification cascade of trophoblast apoptosis/necrosis and oxidative stress, culminating in clinical preeclampsia. This concept immediately leads to a plethora of new and robust therapeutic strategies.
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
