Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Interleukin 1-beta responses to bacterial toxins and sudden infant death syndrome.

FEMS immunology and medical microbiology - 1 Sept 2004

Moscovis Sophia M, Gordon Ann E, Hall Sharron T, Gleeson Maree, Scott Rodney J, Roberts-Thomsom June, Weir Donald M, Busuttil Anthony, Blackwell C Caroline

Abstract excerpt

We tested the hypothesis that significantly higher IL-1beta responses to toxic shock syndrome toxin (TSST) noted for parents of sudden infant death syndrome (SIDS) infants might be due in part to genetic factors such as the IL-1beta (C-511T) and IL-1RN (T+2018C) single nucleotide polymorphisms (S...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Toxins
Share to Enterotoxins
Share to Interleukin-1

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Leukocytes

Share to Racial Groups

Share to Receptors, Interleukin-1

Share to Sudden Infant Death

Share to Superantigens

Source

Identifiers

PMID: 15325407
DOI: 10.1016/j.femsim.2004.06.005

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-15325407-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Interleukin 1-beta responses to bacterial toxins and sudden infant death syndrome.PMID 15325407Read ↗

Interleukin-10 and sudden infant death syndrome.Semantic proximity Read ↗

Evidence for a genetic component in sudden infant death syndrome.Semantic proximity Read ↗

IL6 G-174C associated with sudden infant death syndrome in a Caucasian Australian cohort.Semantic proximity Read ↗

Cytokine responses and sudden infant death syndrome: genetic, developmental, and environmental risk factors.Semantic proximity Read ↗

TNF-alpha and IL-10 gene polymorphisms versus cardioimmunological responses in sudden infant death.Semantic proximity Read ↗

Sudden Infant Death Syndrome and the Genetics of InflammationSemantic proximity Read ↗

Possible pathomechanisms of sudden infant death syndrome: key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants.Semantic proximity Read ↗

Clinical relevance of single nucleotide polymorphisms within the 13 cytokine genes in North Indian trauma hemorrhagic shock patients.Semantic proximity Read ↗

An infectious aetiology of sudden infant death syndrome.Semantic proximity Read ↗

Genetic variant rs16944 in IL1B gene is a risk factor for early-onset sepsis susceptibility and outcome in preterm infants.Semantic proximity Read ↗

A polymorphism in a staphylococcal enterotoxin receptor gene (T cell receptor BV3 recombination signal sequence) is not associated with unexplained sudden unexpected death in infancy in an Australian cohort.Semantic proximity Read ↗

IL-10 gene polymorphisms are associated with infectious cause of sudden infant death.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.