Article
Loss of a callose synthase results in salicylic acid-dependent disease resistance.
Science (New York, N.Y.) - 15 Aug 2003
Nishimura Marc T, Stein Monica, Hou Bi-Huei, Vogel John P, Edwards Herb, Somerville Shauna C
Abstract excerpt
Plants attacked by pathogens rapidly deposit callose, a beta-1,3-glucan, at wound sites. Traditionally, this deposition is thought to reinforce the cell wall and is regarded as a defense response. Surprisingly, here we found that powdery mildew resistant 4 (pmr4), a mutant lacking pathogen-induced callose, became resistant to pathogens, rather than more susceptible. This resistance was due to mutation of a...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.