Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Growth factors, growth factor response elements, and the cardiac phenotype.

Basic research in cardiology - 1 Jan 1992

Schneider M D, McLellan W R, Black F M, Parker T G

Abstract excerpt

Fibroblast growth factors (FGF) and type beta-1 transforming growth factor (TGF beta 1) are pleiotropic regulatory peptides which are expressed in myocardium in a precise developmental and spatial program and are up-regulated, in the adult heart, by ischemia or a hemodynamic burden. The accumulation of trophic factors after aortic banding supports the hypothesis that autocrine or paracrine pathways might function...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Base Sequence
Share to DNA-Binding Proteins
Share to Gene Expression Regulation

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Molecular Sequence Data

Share to Mutation

Share to Nuclear Proteins

Share to Phenotype

Share to Promoter Regions, Genetic

Source

Identifiers

PMID: 1284369
DOI: 10.1007/978-3-642-72477-0_4

Genes

SRF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Growth factors, growth factor response elements, and the cardiac phenotype.PMID 1284369Read ↗

Transforming growth factor-beta in cardiac ontogeny and adaptation.Semantic proximity Read ↗

AFos dissociates cardiac myocyte hypertrophy and expression of the pathological gene program.Semantic proximity Read ↗

Roles of Cardiac Transcription Factors in Cardiac HypertrophySemantic proximity Read ↗

Growth factors, proto-oncogenes, and plasticity of the cardiac phenotype.Semantic proximity Read ↗

Ras-association domain family member 1A (RASSF1A)-where the heart and cancer meet.Semantic proximity Read ↗

Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic responseSemantic proximity Read ↗

SRF-dependent gene expression in isolated cardiomyocytes: Regulation of genes involved in cardiac hypertrophySemantic proximity Read ↗

SRF: a seriously responsible factor in cardiac development and diseaseSemantic proximity Read ↗

Myocardin-Related Transcription Factor A Is a Common Mediator of Mechanical Stress- and Neurohumoral Stimulation-Induced Cardiac Hypertrophic Signaling Leading to Activation of Brain Natriuretic Peptide Gene ExpressionSemantic proximity Read ↗

Signaling pathways mediating cardiac myocyte gene expression in physiological and stress responsesSemantic proximity Read ↗

Regulation of cardiac growth and development by SRF and its cofactors.Semantic proximity Read ↗

Tumor Suppressor Ras-Association Domain Family 1 Isoform A Is a Novel Regulator of Cardiac HypertrophySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.