Article
A gain-of-function mutation in an Arabidopsis Toll Interleukin1 receptor-nucleotide binding site-leucine-rich repeat type R gene triggers defense responses and results in enhanced disease resistance.
The Plant cell - 1 Dec 2002
Shirano Yumiko, Kachroo Pradeep, Shah Jyoti, Klessig Daniel F
Abstract excerpt
In a screen for suppressors of npr1-5-based salicylic acid (SA) insensitivity, we isolated a semidominant gain-of-function mutation, designated ssi4, that confers constitutive expression of several PR (pathogenesis-related) genes, induces SA accumulation, triggers programmed cell death, and enhances resistance to bacterial and oomycete pathogens. Through map-based cloning, ssi4 was identified and found to encode...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.