Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Decay-Accelerating Factor Confers Protection Against Complement-Mediated Podocyte Injury in Acute Nephrotoxic Nephritis

1 May 2002

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chronic Kidney Disease and Diabetes
Share to Complement system in diseases
Share to Renal Diseases and Glomerulopathies

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12003997
DOI: 10.1038/labinvest.3780451

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

Decay-Accelerating Factor Confers Protection Against Complement-Mediated Podocyte Injury in Acute Nephrotoxic NephritisPMID 12003997Read ↗

Transcription factor MafB in podocytes protects against the development of focal segmental glomerulosclerosisSemantic proximity Read ↗

Complement component C5a induces aberrant epigenetic modifications in renal tubular epithelial cells accelerating senescence by Wnt4/βcatenin signaling after ischemia/reperfusion injurySemantic proximity Read ↗

DNA damage is a novel response to sublytic complement C5b-9–induced injury in podocytesSemantic proximity Read ↗

Decay-accelerating factor deficiency increases susceptibility to dextran sulfate sodium-induced colitis: role for complement in inflammatory bowel disease.Semantic proximity Read ↗

Effects of complement factor D deficiency on the renal disease of MRL/lpr miceSemantic proximity Read ↗

The complement system in IgAN: mechanistic context for therapeutic opportunitiesSemantic proximity Read ↗

Kidney diseasesSemantic proximity Read ↗

Complement-Mediated Glomerular Diseases: A Tale of 3 PathwaysSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.