Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enhancement of genetic instability in human B cells by Epstein-Barr virus latent infection.

Mutagenesis - 1 May 2001

Gualandi G, Giselico L, Carloni M, Palitti F, Mosesso P, Alfonsi A M

Abstract excerpt

The level of genetic instability, as assessed by micronucleus (MN) formation, was higher in Epstein-Barr virus (EBV)-converted B-cell lines with one copy of the EBV genome integrated in each cell than in the parental, EBV-negative, B lymphoma cells. MN induced by EBV latency, as analysed by in si...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antioxidants
Share to Glutathione
Share to Herpesvirus 4, Human

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hydrogen Peroxide

Share to Infections

Share to Micronucleus Tests

Share to Oxygen

Share to Peroxides

Source

Identifiers

PMID: 11320144
DOI: 10.1093/mutage/16.3.203

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 31 Aug 2026. Prepared record publication-11320144-2026-08-31.1. Manifest publication-graph-cohort-2026-08-31.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 31 Aug 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enhancement of genetic instability in human B cells by Epstein-Barr virus latent infection.PMID 11320144Read ↗

Epstein–Barr virus latent membrane protein 1 induces micronucleus formation, represses DNA repair and enhances sensitivity to DNA-damaging agents in human epithelial cellsSemantic proximity Read ↗

Epstein–Barr virus lytic replication and cancerSemantic proximity Read ↗

Epstein–Barr virus particles induce centrosome amplification and chromosomal instabilitySemantic proximity Read ↗

Epstein-barr virus-induced resistance to drugs that activate the mitotic spindle assembly checkpoint in Burkitt's lymphoma cells.Semantic proximity Read ↗

Steps Involved in Immortalization and Tumorigenesis in Human B-Lymphoblastoid Cell Lines Transformed by Epstein-Barr VirusSemantic proximity Read ↗

Recurrent chemical reactivations of EBV promotes genome instability and enhances tumor progression of nasopharyngeal carcinoma cellsSemantic proximity Read ↗

Clonal chromosomal aberrations accompanied by strong telomerase activity in immortalization of human B-lymphoblastoid cell lines transformed by Epstein-Barr virusSemantic proximity Read ↗

Epstein-Barr virus induces fragmentation of chromosomal DNA during lytic infectionSemantic proximity Read ↗

Spontaneously occurring micronuclei in infiltrating ductal carcinoma of breast: a potential biomarker for aggressive phenotype detection?Semantic proximity Read ↗

Epstein-Barr Virus Latency in B Cells Leads to Epigenetic Repression and CpG Methylation of the Tumour Suppressor Gene BimSemantic proximity Read ↗

Control of Cell Cycle Entry and Apoptosis in B Lymphocytes Infected by Epstein-Barr VirusSemantic proximity Read ↗

Chromosomal rearrangements after ex vivo Epstein–Barr virus (EBV) infection of human B cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.