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Article

Expression of Na<sup>+</sup>/I<sup>−</sup>Symporter and Pendred Syndrome Genes in Trophoblast Cells<sup>1</sup>

1 Nov 2000

Abstract excerpt

Placental iodide transport is critical for the fetal thyroid function, but the molecular mechanisms of this transport are not understood. The expression of two recently identified iodide transporters, namely the sodium/iodide symporter (NIS) and pendrin, the product of the gene responsible for the Pendred syndrome (PDS), was studied using real-time kinetic quantitative PCR and immunohistochemistry 1) in placental...

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PMID: 11095481
DOI: 10.1210/jcem.85.11.6969

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Expression of Na<sup>+</sup>/I<sup>−</sup>Symporter and Pendred Syndrome Genes in Trophoblast Cells<sup>1</sup>PMID 11095481Read ↗

Expression of Na+/I- Symporter and Pendred Syndrome Genes in Trophoblast CellsSemantic proximity Read ↗

Sodium/Iodide Symporter (NIS) Gene Expression Is the Limiting Step for the Onset of Thyroid Function in the Human FetusSemantic proximity Read ↗

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Pendrin, the Protein Encoded by the Pendred Syndrome Gene (PDS), Is an Apical Porter of Iodide in the Thyroid and Is Regulated by Thyroglobulin in FRTL-5 CellsSemantic proximity Read ↗

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Mutations of the PDS Gene, Encoding Pendrin, Are Associated with Protein Mislocalization and Loss of Iodide Efflux: Implications for Thyroid Dysfunction in Pendred SyndromeSemantic proximity Read ↗

Mutations in the Pendred Syndrome (PDS/SLC26A) Gene: An Increasingly Complex Phenotypic Spectrum From Goiter to Thyroid HypoplasiaSemantic proximity Read ↗

Mutations of the PDS gene, encoding pendrin, are associated with protein mislocalization and loss of iodide efflux: implications for thyroid dysfunction in Pendred syndrome.Semantic proximity Read ↗

The role of pendrin in iodide regulation.Semantic proximity Read ↗

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