Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

gThe Dorothy Russell Memorial Lecture* The molecular and cellular sequelae of experimental traumatic brain injury: pathogenetic mechanisms

1 Aug 1998

Abstract excerpt

The mechanisms underlying secondary or delayed cell death following traumatic brain injury (TBI) are poorly understood. Recent evidence from experimental models of TBI suggest that diffuse and widespread neuronal damage and loss is progressive and prolonged for months to years after the initial insult in selectively vulnerable regions of the cortex, hippocampus, thalamus, striatum, and subcortical nuclei. The...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Mitochondrial Function and Pathology
Share to S100 Proteins and Annexins
Share to Traumatic Brain Injury and Neurovascular Disturbances

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9775390
DOI: 10.1046/j.1365-2990.1998.00121.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

gThe Dorothy Russell Memorial Lecture* The molecular and cellular sequelae of experimental traumatic brain injury: pathogenetic mechanismsPMID 9775390Read ↗

Bench-to-bedside review: Apoptosis/programmed cell death triggered by traumatic brain injury.Semantic proximity Read ↗

Unraveling cell death mechanisms in traumatic brain injury: dynamic roles of ferroptosis and necroptosisSemantic proximity Read ↗

Apoptotic Cell Death Following Traumatic Injury to the Central Nervous SystemSemantic proximity Read ↗

Mild traumatic brain injury induces memory deficits with alteration of gene expression profileSemantic proximity Read ↗

A Review and Rationale for the Use of Genetically Engineered Animals in the Study of Traumatic Brain InjurySemantic proximity Read ↗

Mechanisms of Mitochondrial Oxidative Stress in Brain Injury: From Pathophysiology to TherapeuticsSemantic proximity Read ↗

Genetic and histologic evidence implicates role of inflammation in traumatic brain injury-induced apoptosis in the rat cerebral cortex following moderate fluid percussion injurySemantic proximity Read ↗

TNF alpha and Fas mediate tissue damage and functional outcome after traumatic brain injury in mice.Semantic proximity Read ↗

Traumatic Brain Injury Dysregulates MicroRNAs to Modulate Cell Signaling in Rat HippocampusSemantic proximity Read ↗

Exploring the pathogenesis of traumatic brain injury and traumatic epilepsy based on bioinformatics analysisSemantic proximity Read ↗

Controlled Cortical Impact and Craniotomy Induce Strikingly Similar Profiles of Inflammatory Gene Expression, but with Distinct KineticsSemantic proximity Read ↗

MicroRNAs: The New Challenge for Traumatic Brain Injury DiagnosisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.