[Long QT syndrome: from clinical discovery to molecular etiopathogenesis].

Abstract excerpt

Before the era of Molecular Biology the etiopathogenic mechanism of the long QT Syndrome (LQTS) was hypothetized to be an inhomogeneity in the innervation of the myocardium by the sympathetic system resulting in abnormal myocardial repolarisation, prolongation of the QT interval and various rhythm disorders. The progress of Molecular Biology has led to abandon this hypothesis; it is now agreed that the...

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