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Article

Pathogenicity of a subgroup C feline leukemia virus (FeLV) is augmented when administered in association with certain FeLV recombinants.

Virology - 1 Jan 1994

Mathes L E, Pandey R, Chakrabarti R, Hofman F M, Hayes K A, Stromberg P, Roy-Burman P

Abstract excerpt

There is evidence to suggest that infectious feline leukemia viruses (FeLVs) may be altered biologically because of homologous recombination with non-infectious endogenous FeLV (enFeLV) sequences in the infected cells. To evaluate the role of such recombination events in FeLV pathogenesis, a mole...

Topics

Animals
Animals, Newborn
Antigens, Viral
Base Sequence
Cats
Genetic Variation
Immunohistochemistry
Leukemia Virus, Feline
Molecular Sequence Data
Recombination, Genetic
Retroviridae Infections

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Tumor Virus Infections

Viral Matrix Proteins

Virulence

Source

Identifiers

PMID: 8259654
DOI: 10.1006/viro.1994.1021

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-8259654-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

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Pathogenicity of a subgroup C feline leukemia virus (FeLV) is augmented when administered in association with certain FeLV recombinants.PMID 8259654Read ↗

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