Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The molecular mechanisms in prenatal drug exposure-induced fetal programmed adult cardiovascular disease

20 Apr 2023

Abstract excerpt

The "developmental origins of health and disease" (DOHaD) hypothesis posits that early-life environmental exposures have a lasting impact on individual's health and permanently shape growth, structure, and metabolism. This reprogramming, which results from fetal stress, is believed to contribute to the development of adulthood cardiovascular diseases such as hypertension, coronary artery disease, heart failure,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Fatty Acid Research and Health
Share to Pregnancy and preeclampsia studies

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 37153765
DOI: 10.3389/fphar.2023.1164487

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 23 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The molecular mechanisms in prenatal drug exposure-induced fetal programmed adult cardiovascular diseasePMID 37153765Read ↗

Mechanisms underlying developmental programming of elevated blood pressure and vascular dysfunction: evidence from human studies and experimental animal modelsSemantic proximity Read ↗

Molecular mechanisms underlying the fetal programming of adult diseaseSemantic proximity Read ↗

The programming of cardiovascular diseaseSemantic proximity Read ↗

Developmental toxicity and programming alterations of multiple organs in offspring induced by medication during pregnancySemantic proximity Read ↗

The highs and lows of programmed cardiovascular disease by developmental hypoxia: studies in the chicken embryoSemantic proximity Read ↗

The molecular mechanisms of IUGR programmed adulthood cardiovascular diseaseSemantic proximity Read ↗

Environmental Alterations during Embryonic Development: Studying the Impact of Stressors on Pluripotent Stem Cell-Derived CardiomyocytesSemantic proximity Read ↗

A review on the genetic, environmental, and lifestyle aspects of the early-life origins of cardiovascular disease.Semantic proximity Read ↗

Prenatal stress, glucocorticoids and the programming of adult diseaseSemantic proximity Read ↗

Animal Models That Elucidate Basic Principles of the Developmental Origins of Adult DiseasesSemantic proximity Read ↗

Developmental origins of health and disease: environmental exposures.Semantic proximity Read ↗

Developmental toxicant exposures and sex-specific effects on epigenetic programming and cardiovascular health across generationsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.