Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mechanism of apoptosis activation by Curcumin rescued mutant p53Y220C in human pancreatic cancer

22 Aug 2022

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Curcumin's Biomedical Applications
Share to RNA Interference and Gene Delivery

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 36007676
DOI: 10.1016/j.bbamcr.2022.119343

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mechanism of apoptosis activation by Curcumin rescued mutant p53Y220C in human pancreatic cancerPMID 36007676Read ↗

Control of Apoptosis in Treatment and Biology of Pancreatic CancerSemantic proximity Read ↗

Autophagy induced by p53-reactivating molecules protects pancreatic cancer cells from apoptosisSemantic proximity Read ↗

Autophagy, p53, and pancreatic cancer.Semantic proximity Read ↗

Apoptosis pathways and their therapeutic exploitation in pancreatic cancerSemantic proximity Read ↗

Apoptosis: targets in pancreatic cancer.Semantic proximity Read ↗

Resveratrol mediated the proliferation and apoptosis of gastric cancer cells by modulating the PI3K/Akt/P53 signaling pathwaySemantic proximity Read ↗

Targeting of cancer cell death mechanisms by resveratrol: a reviewSemantic proximity Read ↗

Wild type and gain of function mutant TP53 can regulate the sensitivity of pancreatic cancer cells to chemotherapeutic drugs, EGFR/Ras/Raf/MEK, and PI3K/mTORC1/GSK-3 pathway inhibitors, nutraceuticals and alter metabolic properties.Semantic proximity Read ↗

Mutant p53 stimulates chemoresistance of pancreatic adenocarcinoma cells to gemcitabine.Semantic proximity Read ↗

Apoptosome dysfunction in human cancerSemantic proximity Read ↗

In silico and in vitro analysis of coumarin derivative induced anticancer effects by undergoing intrinsic pathway mediated apoptosis in human stomach cancerSemantic proximity Read ↗

Curcumin Rescues a PINK1 Knock Down SH-SY5Y Cellular Model of Parkinson’s Disease from Mitochondrial Dysfunction and Cell DeathSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.