Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

TFF1 Induces Aggregation and Reduces Motility of Helicobacter pylori

12 Feb 2021

Abstract excerpt

Gastric cancer is considered one of the most common malignancies in humans and Helicobacter pylori infection is the major environmental risk factor of gastric cancer development. Given the high spread of this bacterium whose infection is mostly asymptomatic, H. pylori colonization persists for a long time, becoming chronic and predisposing to malignant transformation. The first defensive barrier from bacterial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Galectins and Cancer Biology
Share to Helicobacter pylori-related gastroenterology studies
Share to Veterinary medicine and infectious diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33673347
DOI: 10.3390/ijms22041851

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

TFF1 Induces Aggregation and Reduces Motility of Helicobacter pyloriPMID 33673347Read ↗

How H elicobacter pylori senses, targets and interacts with the gastric epitheliumSemantic proximity Read ↗

Streptococcus mitis Induces Conversion of Helicobacter pylori to Coccoid Cells during Co-Culture In VitroSemantic proximity Read ↗

Alterations in Helicobacter pylori Triggered by Contact with Gastric Epithelial CellsSemantic proximity Read ↗

Adherens junctions as targets of microorganisms: A focus on <i>Helicobacter pylori</i>Semantic proximity Read ↗

Role for α- l -fucosidase in the control of Helicobacter pylori -infected gastric cancer cellsSemantic proximity Read ↗

BabA dependent binding of Helicobacter pylori to human gastric mucins cause aggregation that inhibits proliferation and is regulated via ArsSSemantic proximity Read ↗

Human Gastric Mucins Differently Regulate Helicobacter pylori Proliferation, Gene Expression and Interactions with Host CellsSemantic proximity Read ↗

Myricetin as an Antivirulence Compound Interfering with a Morphological Transformation into Coccoid Forms and Potentiating Activity of Antibiotics against Helicobacter pyloriSemantic proximity Read ↗

The trefoil factor interacting protein TFIZ1 binds the trefoil protein TFF1 preferentially in normal gastric mucosal cells but the co-expression of these proteins is deregulated in gastric cancerSemantic proximity Read ↗

The impact of the flagellar protein gene fliK on Helicobacter pylori biofilm formationSemantic proximity Read ↗

The Helicobacter pylori Autotransporter ImaA Tempers the Bacterium's Interaction with α5β1 Integrin.Semantic proximity Read ↗

Trefoil Factor Family (TFF) Peptides and their Different Roles in the Mucosal Innate Immune Defense and More: An UpdateSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.