Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

SALL4 suppresses reactive oxygen species in pancreatic ductal adenocarcinoma phenotype via FoxM1/Prx III axis.

Biochemical and biophysical research communications - 18 Sept 2018

Huynh Do Luong, Zhang Jiao Jiao, Chandimali Nisansala, Ghosh Mrinmoy, Gera Meeta, Kim Nameun, Park Yang Ho, Kwon Taeho, Jeong Dong Kee

Abstract excerpt

Pancreatic ductal adenocarcinoma (PDAC) is a major malignant phenotype in pancreatic cancer, which is one of the most death causes by cancer in the world. PDAC developed from pancreatic intra-epithelial neoplasms (PanINs) and poorly diagnosed at early stages. Beside of high drug resistance, metastasis is the great concern during pancreatic cancer treatment. SALL4 expression is inherent in the upregulations of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carcinoma, Pancreatic Ductal
Share to Cell Movement

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cell Proliferation

Share to Cell Transdifferentiation

Share to Neoplasm Metastasis

Share to Phenotype

Share to Reactive Oxygen Species

Share to Transcription Factors

Source

Identifiers

PMID: 29958885
DOI: 10.1016/j.bbrc.2018.06.145

Genes

SALL4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

SALL4 suppresses reactive oxygen species in pancreatic ductal adenocarcinoma phenotype via FoxM1/Prx III axis.PMID 29958885Read ↗

Potential Prognostic Biomarkers of OSBPL Family Genes in Patients with Pancreatic Ductal AdenocarcinomaSemantic proximity Read ↗

Unraveling the role of Major Vault Protein as a novel immune-related biomarker that promotes the proliferation and migration in pancreatic adenocarcinomaSemantic proximity Read ↗

Comprehensive Analysis on Prognosis and Immune Infiltration of Lysyl Oxidase Family Members in Pancreatic Adenocarcinoma With Experimental VerificationSemantic proximity Read ↗

MKLN1-AS promotes pancreatic cancer progression as a crucial downstream mediator of HIF-1α through miR-185-5p/TEAD1 pathwaySemantic proximity Read ↗

Oxysterol-Binding Protein 2 Promotes Pancreatic Ductal Adenocarcinoma Progression Through Epithelial-Mesenchymal Transition ProcessSemantic proximity Read ↗

LOX and ACSL5 as potential relapse markers for pancreatic cancer patientsSemantic proximity Read ↗

Repurposing DPP-4 inhibitors as anticancer agents in KRAS-mutated pancreatic ductal adenocarcinoma.Semantic proximity Read ↗

Role of bio‐flavonols and their derivatives in improving mitochondrial dysfunctions associated with pancreatic tumorigenesisSemantic proximity Read ↗

LINC02086 inhibits ferroptosis and promotes malignant phenotypes of pancreatic cancer via miR-342-3p/CA9 axis.Semantic proximity Read ↗

Epigenetic Alterations in Pancreatic Cancer Metastasis.Semantic proximity Read ↗

Targeting reactive oxygen species in development and progression of pancreatic cancer.Semantic proximity Read ↗

ALDOA functions as an oncogene in the highly metastatic pancreatic cancerSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.