Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Upregulation of miR-483-3p contributes to endothelial progenitor cells dysfunction in deep vein thrombosis patients via SRF

22 Jan 2016

Abstract excerpt

BACKGROUND: Endothelial progenitor cells (EPCs) contribute to recanalization of deep vein thrombosis (DVT). This study aimed to detect miRNA expression profiles in EPCs from patients with DVT and characterize the role of miRNA in EPCs dysfunction. METHODS: EPCs was isolated from DVT patients and control subjects, and miRNA expression profiles were compared to screen differential miRNAs. The candidate miRNAs were...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angiogenesis and VEGF in Cancer
Share to Pulmonary Hypertension Research and Treatments
Share to Venous Thromboembolism Diagnosis and Management

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26801758
DOI: 10.1186/s12967-016-0775-2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Upregulation of miR-483-3p contributes to endothelial progenitor cells dysfunction in deep vein thrombosis patients via SRFPMID 26801758Read ↗

Non-coding RNAs regulating endothelial progenitor cells for venous thrombosis: promising therapy and innovationSemantic proximity Read ↗

miR-21 induces endothelial progenitor cells proliferation and angiogenesis via targeting FASLG and is a potential prognostic marker in deep venous thrombosisSemantic proximity Read ↗

Advances in microRNA regulation of deep vein thrombosis through venous vascular endothelial cells (Review)Semantic proximity Read ↗

Upregulated miR-206 Aggravates Deep Vein Thrombosis by Regulating GJA1-Mediated Autophagy of Endothelial Progenitor CellsSemantic proximity Read ↗

Circulating microRNA expression and their target genes in deep vein thrombosisSemantic proximity Read ↗

MicroRNA-181b-5p/HEY2 axis is involved in the progress of deep venous thrombosis via mediating vascular endothelial injurySemantic proximity Read ↗

Clinical significance and underlying mechanism of long non‐coding RNA SNHG12 in lower extremity deep venous thrombosisSemantic proximity Read ↗

Human cancer cells suppress behaviors of endothelial progenitor cells through miR-21 targeting IL6R.Semantic proximity Read ↗

MiR‐9 promotes angiogenesis of endothelial progenitor cell to facilitate thrombi recanalization via targeting TRPM7 through PI3K/Akt/autophagy pathwaySemantic proximity Read ↗

The role of microRNAs disturbing endothelial function involved in the pathogenesis of thrombosis and thrombolysisSemantic proximity Read ↗

Downregulation of miR-103a-3p Contributes to Endothelial Progenitor Cell Dysfunction in Deep Vein Thrombosis Through PTEN TargetingSemantic proximity Read ↗

Biomarkers for the (cancer-related) risk of thrombosis in the veins: microRNAsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.