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Article

Emergence of a BCR-ABL translocation in a patient with the JAK2V617F mutation: evidence for secondary acquisition of BCR-ABL in the JAK2V617F clone.

Journal of clinical oncology : official journal of the American Society of Clinical Oncology - 20 Jul 2014

Yamada Osamu, Mahfoudhi Emna, Plo Isabelle, Ozaki Kohji, Nakatake Mayuka, Akiyama Masaharu, Yamada Hisashi, Kawauchi Kiyotaka, Vainchenker William

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Share to Aged
Share to Fusion Proteins, bcr-abl
Share to BCR gene

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Janus Kinase 2

Share to Leukemia, Lymphocytic, Chronic, B-Cell

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Share to Translocation, Genetic

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Identifiers

PMID: 24516035
DOI: 10.1200/JCO.2012.47.8669

Genes

BCR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

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Emergence of a BCR-ABL translocation in a patient with the JAK2V617F mutation: evidence for secondary acquisition of BCR-ABL in the JAK2V617F clone.PMID 24516035Read ↗

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BCR-ABL1-positive and JAK2 V617F-positive clones in 23 patients with both aberrations reveal biologic and clinical importance.Semantic proximity Read ↗

BCR/ABL-negative chronic myeloproliferative disorders: JAK2 mutation and beyond.Semantic proximity Read ↗

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The saga of JAK2 mutations and translocations in hematologic disorders: pathogenesis, diagnostic and therapeutic prospects, and revised World Health Organization diagnostic criteria for myeloproliferative neoplasmsSemantic proximity Read ↗

Evidence suggesting the presence of a stem cell clone anteceding the acquisition of the JAK2-V617F mutation.Semantic proximity Read ↗

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