Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Strigolactone/MAX2-induced degradation of brassinosteroid transcriptional effector BES1 regulates shoot branching.

Developmental cell - 23 Dec 2013

Wang Yuan, Sun Shiyong, Zhu Wenjiao, Jia Kunpeng, Yang Hongquan, Wang Xuelu

Abstract excerpt

Strigolactones (SLs), a class of the most recently identified terpenoid phytohormones, play essential roles in plant development, specifically in suppressing shoot branching. MAX2, a subunit of an SCF E3 ligase and a positive regulator that inhibits shoot branching, is likely a key SL signaling component. Here, we provide genetic and biochemical evidence to demonstrate that BES1 interacts with MAX2 and acts as...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brassinosteroids
Share to Fluorescence
Share to Image Processing, Computer-Assisted

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Plant Growth Regulators

Share to Plant Shoots

Share to Proteolysis

Share to Ubiquitin

Share to Ubiquitination

Source

Identifiers

PMID: 24369836
DOI: 10.1016/j.devcel.2013.11.010

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Strigolactone/MAX2-induced degradation of brassinosteroid transcriptional effector BES1 regulates shoot branching.PMID 24369836Read ↗

Strigolactone Signaling in Arabidopsis Regulates Shoot Development by Targeting D53-Like SMXL Repressor Proteins for Ubiquitination and Degradation.Semantic proximity Read ↗

The strigolactone receptor DWARF14 regulates flowering time in Arabidopsis.Semantic proximity Read ↗

Strigolactone signaling regulates cambial activity through repression of WOX4 by transcription factor BES1.Semantic proximity Read ↗

Knockdown of strigolactone biosynthesis genes in Populus affects BRANCHED1 expression and shoot architecture.Semantic proximity Read ↗

A role for more axillary growth1 (MAX1) in evolutionary diversity in strigolactone signaling upstream of MAX2.Semantic proximity Read ↗

Strigolactones enhance competition between shoot branches by dampening auxin transport.Semantic proximity Read ↗

DWARF3 participates in an SCF complex and associates with DWARF14 to suppress rice shoot branching.Semantic proximity Read ↗

BES1 accumulates in the nucleus in response to brassinosteroids to regulate gene expression and promote stem elongation.Semantic proximity Read ↗

Strigolactone signalling inhibits trehalose 6-phosphate signalling independently of BRC1 to suppress shoot branching.Semantic proximity Read ↗

Strigolactones and Brassinosteroids Antagonistically Regulate the Stability of the D53-OsBZR1 Complex to Determine FC1 Expression in Rice Tillering.Semantic proximity Read ↗

SMAX1-LIKE/D53 Family Members Enable Distinct MAX2-Dependent Responses to Strigolactones and Karrikins in Arabidopsis.Semantic proximity Read ↗

d14, a strigolactone-insensitive mutant of rice, shows an accelerated outgrowth of tillers.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.