Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Overproduction of chl B retards senescence through transcriptional reprogramming in Arabidopsis.

Plant & cell physiology - 1 Mar 2012

Sakuraba Yasuhito, Balazadeh Salma, Tanaka Ryouichi, Mueller-Roeber Bernd, Tanaka Ayumi

Abstract excerpt

Leaf senescence is a developmentally and environmentally regulated process which includes global changes in gene expression. Using Arabidopsis as a model, we modified Chl arrangement in photosystems by overexpressing the catalytic domain (the C domain) of chlorophyllide a oxygenase (CAO) fused with the linker domain (the B domain) of CAO and green fluorescent protein (GFP). In these plants (referred to as the BCG...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arabidopsis Proteins
Share to Chlorophyll

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cluster Analysis

Share to Darkness

Share to Green Fluorescent Proteins

Share to Oxygenases

Share to Photosynthesis

Share to Plant Leaves

Source

Identifiers

PMID: 22285931
DOI: 10.1093/pcp/pcs006

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Overproduction of chl B retards senescence through transcriptional reprogramming in Arabidopsis.PMID 22285931Read ↗

Suppressor of Overexpression of CO 1 Negatively Regulates Dark-Induced Leaf Degreening and Senescence by Directly Repressing Pheophytinase and Other Senescence-Associated Genes in Arabidopsis.Semantic proximity Read ↗

Light intensity-dependent modulation of chlorophyll b biosynthesis and photosynthesis by overexpression of chlorophyllide a oxygenase in tobacco.Semantic proximity Read ↗

The N-terminal domain of chlorophyllide a oxygenase confers protein instability in response to chlorophyll B accumulation in Arabidopsis.Semantic proximity Read ↗

The NAC transcription factor ANAC046 is a positive regulator of chlorophyll degradation and senescence in Arabidopsis leaves.Semantic proximity Read ↗

The control of chlorophyll catabolism and the status of yellowing as a biomarker of leaf senescence.Semantic proximity Read ↗

Arabidopsis thylakoid formation 1 is a critical regulator for dynamics of PSII-LHCII complexes in leaf senescence and excess light.Semantic proximity Read ↗

PGL, encoding chlorophyllide a oxygenase 1, impacts leaf senescence and indirectly affects grain yield and quality in rice.Semantic proximity Read ↗

Manipulation of a Senescence-Associated Gene Improves Fleshy Fruit Yield.Semantic proximity Read ↗

Age-triggered and dark-induced leaf senescence require the bHLH transcription factors PIF3, 4, and 5.Semantic proximity Read ↗

Overexpression of the CBF2 transcriptional activator in Arabidopsis delays leaf senescence and extends plant longevity.Semantic proximity Read ↗

The Non-Mendelian Green Cotyledon Gene in Soybean Encodes a Small Subunit of Photosystem II.Semantic proximity Read ↗

Pheophorbide a May Regulate Jasmonate Signaling during Dark-Induced Senescence.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.