Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Transcriptional profiling of high pigment-2dg tomato mutant links early fruit plastid biogenesis with its overproduction of phytonutrients.

Plant physiology - 1 Oct 2007

Kolotilin Igor, Koltai Hinanit, Tadmor Yaakov, Bar-Or Carmiya, Reuveni Moshe, Meir Ayala, Nahon Sahadia, Shlomo Haviva, Chen Lea, Levin Ilan

Abstract excerpt

Phenotypes of the tomato (Solanum lycopersicum) high pigment-2(dg) (hp-2(dg)) and hp-2(j) mutants are caused by lesions in the gene encoding DEETIOLATED1, a negative regulator of light signaling. Homozygous hp-2(dg) and hp-2(j) plants display a plethora of distinctive developmental and metabolic phenotypes in comparison to their normal isogenic counterparts. These mutants are, however, best known for the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carotenoids
Share to Fruit

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Gene Expression Profiling

Share to Gene Expression Regulation, Plant

Share to Plant Proteins

Share to Plastids

Share to Solanum lycopersicum

Share to Transcription, Genetic

Source

Open in PubMed Open full text

Identifiers

PMID: 17704236
DOI: 10.1104/pp.107.102962
PMCID: PMC2048735

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transcriptional profiling of high pigment-2dg tomato mutant links early fruit plastid biogenesis with its overproduction of phytonutrients.PMID 17704236Read ↗

The light-hyperresponsive high pigment-2dg mutation of tomato: alterations in the fruit metabolome.Semantic proximity Read ↗

Characterization of the intense pigment tomato genotype emphasizing targeted fruit metabolites and chloroplast biogenesis.Semantic proximity Read ↗

Carotenoid profiling and biosynthetic gene expression in flesh and peel of wild-type and hp-1 tomato fruit under UV-B depletion.Semantic proximity Read ↗

Increases in cell elongation, plastid compartment size and phytoene synthase activity underlie the phenotype of the high pigment-1 mutant of tomato.Semantic proximity Read ↗

Chromoplast-specific carotenoid-associated protein appears to be important for enhanced accumulation of carotenoids in hp1 tomato fruits.Semantic proximity Read ↗

Response of wild-type and high pigment-1 tomato fruit to UV-B depletion: flavonoid profiling and gene expression.Semantic proximity Read ↗

An orange ripening mutant links plastid NAD(P)H dehydrogenase complex activity to central and specialized metabolism during tomato fruit maturation.Semantic proximity Read ↗

Phenotype of the tomato high pigment-2 mutant is caused by a mutation in the tomato homolog of DEETIOLATED1.Semantic proximity Read ↗

Altered chloroplast development and delayed fruit ripening caused by mutations in a zinc metalloprotease at the lutescent2 locus of tomato.Semantic proximity Read ↗

Overexpression of UV-DAMAGED DNA BINDING PROTEIN 1 links plant development and phytonutrient accumulation in high pigment-1 tomato.Semantic proximity Read ↗

Skin colour, carotenogenesis and chlorophyll degradation mutant alleles: genetic orchestration behind the fruit colour variation in tomato.Semantic proximity Read ↗

Uniform ripening encodes a Golden 2-like transcription factor regulating tomato fruit chloroplast development.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.