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Article

ras2 Controls morphogenesis, pheromone response, and pathogenicity in the fungal pathogen Ustilago maydis.

Eukaryotic cell - 1 Dec 2002

Lee Nancy, Kronstad James W

Abstract excerpt

Ustilago maydis, a pathogen of maize, is a useful model for the analysis of mating, pathogenicity, and the morphological transition between budding and filamentous growth in fungi. As in other fungi, these processes are regulated by conserved signaling mechanisms, including the cyclic AMP (cAMP)/protein kinase A (PKA) pathway and at least one mitogen-activated protein kinase (MAP kinase) pathway. A current...

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Source

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Identifiers

PMID: 12477796
DOI: 10.1128/EC.1.6.954-966.2002
PMCID: PMC138767

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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ras2 Controls morphogenesis, pheromone response, and pathogenicity in the fungal pathogen Ustilago maydis.PMID 12477796Read ↗

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The PAK family kinase Cla4 is required for budding and morphogenesis in Ustilago maydis.Semantic proximity Read ↗

Lipid-induced filamentous growth in Ustilago maydis.Semantic proximity Read ↗

cAMP regulates morphogenesis in the fungal pathogen Ustilago maydis.Semantic proximity Read ↗

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Calcineurin is an antagonist to PKA protein phosphorylation required for postmating filamentation and virulence, while PP2A is required for viability in Ustilago maydis.Semantic proximity Read ↗

Serial analysis of gene expression reveals conserved links between protein kinase A, ribosome biogenesis, and phosphate metabolism in Ustilago maydis.Semantic proximity Read ↗

Activation of the cAMP pathway in Ustilago maydis reduces fungal proliferation and teliospore formation in plant tumors.Semantic proximity Read ↗

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